succinate/hemoragie

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Activation of hypoxia-inducible factor -1α via succinate dehydrogenase pathway during acute lung injury induced by trauma/hemorrhagic shock.

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Hypoxia-inducible factor (HIF)-1α is a transcription factor that is critical for tissue adaption to hypoxia and inflammation. Previous studies had indicated that normoxic activation of HIF-1α in cancer involves inhibition or mutation of the metabolic enzyme succinate dehydrogenase (SDH). We have

Methylprednisolone sodium succinate reduces BBB disruption and inflammation in a model mouse of intracranial haemorrhage.

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Inflammation and disruption of the blood-brain barrier (BBB) cause oedema and secondary brain injury after intracranial haemorrhage (ICH), which is closely related to patient prognosis. Methylprednisolone sodium succinate (MPSS), a well-known immunosuppressive agent, is widely applied in many

All animals are equal but some animals are more equal than others: Plasma lactate and succinate in hemorrhagic shock: A comparison in rodents, swine, non-human primates and injured patients.

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BACKGROUND Plasma levels of lactate and succinate are predictors of mortality in critically injured patients in military and civilian settings. In relative terms, these metabolic derangements have been recapitulated in rodent, swine, and nonhuman primate models of severe hemorrhage. However, no

Effects of lactated Ringer solution and prednisolone sodium succinate on dogs with induced hemorrhagic shock.

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Hemorrhagic shock was induced in nonsplenectomized dogs by removing 41% of their blood volume over a 15-minute period. Hemodynamic and metabolic variables were determined prior to and for 3 hours after completion of hemorrhage. One group of 5 dogs was not treated. After the 30-minute sample was

Gastric hemorrhage in dogs given high doses of methylprednisolone sodium succinate.

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OBJECTIVE To determine whether healthy dogs given high doses of methylprednisolone sodium succinate (MPSS) develop gastrointestinal tract ulcers and hemorrhage. METHODS 19 healthy male hound-type dogs. METHODS Dogs were assigned randomly to intravenously receive high doses of MPSS (30 mg/kg of body

[Comparative evaluation of the effects of crystalloid solutions containing sodium lactate and sodium succinate on the hemodynamic indicators in different types of hemorrhage].

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The results are presented of the hemodynamics investigations under the influence of two saline infusion solutions in varying types of hemorrhage. The suggested crystalloid solution succinasol contains sodium succinate (instead of lactate that is present in lactasol) which improves energetic

Efficacy of misoprostol in prevention of gastric hemorrhage in dogs treated with high doses of methylprednisolone sodium succinate.

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OBJECTIVE To determine whether administration of misoprostol prevents gastric hemorrhage in healthy dogs treated with high doses of methylprednisolone sodium succinate (MPSS). METHODS 18 healthy hound-type dogs of both sexes. METHODS All dogs were given high doses of MPSS (30 mg/kg of body weight,

Pharmacokinetics of methylprednisolone succinate, methylprednisolone, and lidocaine in the normal dog and during hemorrhagic shock.

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Pharmacokinetics of methylprednisolone succinate and methylprednisolone following methylprednisolone sodium succinate administration were studied in five dogs under normal conditions and then during a severe hemorrhagic shock. In order to evaluate hepatic blood flow, lidocaine clearance was

Glutamine metabolism drives succinate accumulation in plasma and the lung during hemorrhagic shock.

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Metabolomic investigations have consistently reported succinate accumulation in plasma after critical injury. Succinate receptors have been identified on numerous tissues, and succinate has been directly implicated in postischemic inflammation, organ dysfunction, platelet activation, and the

[Alteration of thrombocytopenic hemorrhages with estriol succinate].

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The effect of oestriol succinate on bleeding time.

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[ESTRIOL SUCCINATE IN THE TREATMENT OF HEMORRHAGIC DIATHESES].

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Haemorrhagic mechanisms of some snake venoms in relation to protection by estriol succinate of blood vessel damage.

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[CLINICAL EXPERIENCES WITH ESTRIOL SUCCINATE IN THE TREATMENT OF HEMORRHAGIC DIATHESIS IN INTERNAL MEDICINE, OBSTETRICS AND GYNECOLOGY].

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INHIBITORY EFFECTS OF ESTRIOL-16,17-DISODIUM SUCCINATE ON LOCAL HAEMORRHAGES INDUCED BY SNAKE VENOM IN CANINE HEART-LUNG PREPARATIONS.

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