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sulfuric acid/edema

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ArticoleStudii cliniceBrevete
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[Case of toxic pulmonary edema developed after total action of vapors of hydrofluoric, nitrous and sulfuric acid mixture].

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Accidental fatal inhalation of sulfuric acid fumes.

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A man found dead with circumstantial evidence indicating exposure to sulfuric acid vapor. He had been applying a solution of concentrated sulfuric acid to a drain pipe in an attempt to clear a blockage. External examination showed moderate corrosive damage around his mouth and nose, on his forearms

Adult respiratory distress syndrome from sulfuric acid fume inhalation.

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A 23-year-old man had adult respiratory distress syndrome (ARDS) caused by acute exposure to sulfuric acid fumes. The patient survived the initial hospitalization to be readmitted later with a lung abscess. After therapy, his chest roentgenogram and pulmonary function tests revealed no abnormalities

Extensive multi-organ necrosis secondary to sulfuric acid ingestion.

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This is a report of a case in which a significant quantity of sulfuric acid was ingested, causing life threatening respiratory distress, secondary to glottic injury and edema, and cardiovascular collapse. Vigorous resuscitive efforts allowed survival through the first 48 hours and two major

Respiratory tract lesions in guinea pigs exposed to sulfuric acid mist.

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Guinea pigs were exposed inhalation chambers to 25 mg/m3 sulfuric acid mist 6 h/d for 2 d, and the acute respiratory effects were correlated by light and electron microscopy. This concentration of acid was selected since lower concentrations result in only slight effects while higher concentrations

A fatality caused by accidental production of hydrogen sulfide.

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A 55-year-old male Caucasian truck driver was dead at the scene after breathing hydrogen sulfide (H(2)S) produced by an accidental transfer of sodium hydrogen sulfide (NaHS) from a tanker truck to a tank containing 4% sulfuric acid (H(2)SO(4)) and iron(II) sulfate (FeSO(4)). Autopsy of the

He-Ne laser therapy by fibroendoscopy in the mucosa of the equine upper airway.

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OBJECTIVE A study was made of the effects of low-level laser irradiation on the cicatrization of superficial wounds in the pharyngeal mucosa of the horse. METHODS Duplicate pharyngeal mucosal ulcers were induced in 12 Spanish horses with submucosal injection of sulfuric acid. A fibroendoscope and an

Ventilatory responses in awake guinea pigs exposed to acid aerosols.

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This study reports experiments designed to evaluate the dose and temporal effects of an atmospheric pollutant, sulfuric acid (H2SO4) aerosol, on the dynamic components of the respiratory cycle. Ventilation was measured in a whole-body barometric plethysmograph in unanesthetized, unrestrained animals

Bilateral pneumothorax following acute inhalation injury.

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BACKGROUND Inhalation injury can be thermal and/or chemical. We report bilateral pneumothorax following acute inhalation injury. METHODS A male worker in an upholstery factory was confined in the tanning machine for 15 min. The device was used to contain sodium sulfate, sulfur dioxide, and sulfuric

Corneal epithelial regeneration and adhesion following acid burns in the rhesus monkey.

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For study of corneal adhesion after chemical injury, sulfuric acid was used to burn the central and peripheral corneal epithelium in rhesus monkeys. The adhesion properties of the regenerating epithelium were sequentially followed by clinical assessment and by serial corneal biopsy specimens

Amyloidosis in six dairy cows.

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Amyloidosis was diagnosed in 6 Holstein cows that were examined because of chronic intractable diarrhea. Besides diarrhea, the chief finding was a nephrotic-like syndrome, in that there was edema, hypoproteinemia, and proteinuria. Other consistent clinicopathologic abnormalities were

pH effects on ciliomotility and morphology of respiratory mucosa.

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Tracheal cilia of cows exposed, in vitro, for 20 hours to different acidities and alkalinities of sulfuric acid and sodium hydroxide, respectively, showed ciliomotility at pH values as low as 4.9, while the epithelial cells began to be expelled from the mucosa at pH 6.7, initiating a reduction of

Wound healing activity of NOE-aspirin: a pre-clinical study.

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Aspirin, one of the oldest non-steroidal anti-inflammatory drugs, impedes tissue repair by virtue of retarding inflammation. The present study was undertaken to find out if linking of nitrooxyethyl ester to aspirin reverses its healing-depressant propensity. Nitrooxyethyl ester of aspirin (NOE-Asp)
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