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Glucocorticoid status affects the response of rat striatal dopamine D2 receptors to hyperthermia and turpentine treatment.

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The influence of glucocorticoid status of the rats (absence of glucocorticoid hormones achieved by adrenalectomy and substitution by dexamethasone 5 mg/kg b.w.) on the response of striatal dopamine-D2 receptors to environmentally induced hyperthermia and treatment with a local inflammatory agent

Attenuated fever in rats during late pregnancy is linked to suppressed interleukin-6 production after localized inflammation with turpentine.

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An attenuated fever response to pathogens during late pregnancy is a phenomenon that has been described in several mammalian species, and although mechanisms are not completely understood, decreased prostaglandin E2 (PGE2) synthesis has been implicated. Upstream of PGE2, there is evidence to suggest

Production of mice deficient in genes for interleukin (IL)-1alpha, IL-1beta, IL-1alpha/beta, and IL-1 receptor antagonist shows that IL-1beta is crucial in turpentine-induced fever development and glucocorticoid secretion.

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Interleukin (IL)-1 is a major mediator of inflammation and exerts pleiotropic effects on the neuro-immuno-endocrine system. To elucidate pathophysiological roles of IL-1, we have first produced IL-1alpha/beta doubly deficient (KO) mice together with mice deficient in either the IL-1alpha, IL-1beta,

Effect of turpentine-induced fever during the enamel formation of rat incisor.

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OBJECTIVE Some epidemiological studies have indicated that diseases resulting in prolonged and sustained fever, such as exanthemata, respiratory infections and otitis media in infantile period or childhood are likely to have a marked deleterious effect on enamel formation, but the relationship

On Spirit of Turpentine in Puerperal Fever.

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On the Use of Turpentine in Puerperal Fever.

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TURPENTINE ENEMATA IN THE TREATMENT OF ENTERIC FEVER.

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On Spirit of Turpentine in the Yellow Fever.

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Case of Puerperal Fever, Successfully Treated by Oil of Turpentine.

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Observations on the Good Effects of Oil of Turpentine in Puerperal Fever.

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Case of Enteric Fever, Showing the Value of Turpentine in the Treatment of This Disease.

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On the External Application of Oil of Turpentine as a Substitute for Quinine in Intermittent Fever, with Reports of Cases.

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Intracerebroventricular injection of neuronal and inducible nitric oxide synthase inhibitors does not influence febrile response in rats during turpentine abscess.

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The purpose of this study was to investigate the role of neuronal nitric oxide synthase (nNOS) and inducible NOS (iNOS) in the brain during development of fever in response to localized tissue inflammation caused by injection of turpentine in freely moving biotelemetered rats. To determine the role

Tumor necrosis factor-alpha and fever after peripheral inflammation in the rat.

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The involvement of endogenous tumor necrosis factor-alpha (TNF-alpha) in the pyrogenic [i.e., rise in colonic temperature (Tc)] and thermogenic [increase in oxygen consumption (VO2)] responses to inflammation was investigated in rats subjected to an intramuscular injection of turpentine. Turpentine

[Poisoning by spirits of turpentine or turpentine oil. Review of its treatment apropos of a personal case].

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Authors deduce from their experience during 1979 that intoxication by spirit oil of turpentine is an accident with few repercussions in infancy, because the quantity ingested does not reach toxic levels, except for children over seven who try to commit suicide. Gastric lavage must not be practiced

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