antidepressants/инфаркт

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Trends in the prescribing of antidepressants following acute myocardial infarction, 1993-2002.

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OBJECTIVE There has been a substantial increase in the prescribing of antidepressants on a population basis and in particular serotonin reuptake inhibitors (SSRIs). SSRIs have lower cardiac toxicity than tricyclic antidepressants (TCAs). We examined how the prescribing of antidepressants to patients

Use of antidepressants and the risk of myocardial infarction in middle-aged and older adults: a matched case-control study.

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OBJECTIVE Antidepressants, specifically selective serotonin reuptake-inhibiting antidepressants (SSRIs), decrease platelet activation and aggregation in in vitro experiments and could therefore decrease the risk of myocardial infarction (MI). However, prior studies addressing this hypothesis showed

Impact of antidepressants use on risk of myocardial infarction: A systematic review and meta-analysis.

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OBJECTIVE The aim of the study was to perform a systematic review and meta-analysis to determine the association between antidepressants use and risk of myocardial infarction (MI), and whether this association differs between tricyclic antidepressants (TCAs) and selective serotonin reuptake

Two cases of emotional disorder after middle cerebral artery infarction showing distinct responses to antidepressant treatment.

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Many emotional disturbances such as post-stroke depression (PSD) and emotional incontinence (EI) commonly occur following cerebrovascular events. The efficacy of antidepressants for these conditions has been established but their comorbid treatment has not been well characterized. In the current

Use of antidepressants and risk of hospitalization for acute myocardial infarction: A nationwide case-crossover study.

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BACKGROUND We aimed to investigate the association between antidepressant use and the risk of hospitalization for acute myocardial infarction (AMI). METHODS A case-crossover study was conducted using a nationwide population-based sample from Taiwan's National Health Insurance Research Database. A

DIFFERENTIAL EFFECTS OF ANTIDEPRESSANT SUBGROUPS ON RISK OF ACUTE MYOCARDIAL INFARCTION: A NESTED CASE-CONTROL STUDY.

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The primary objective of this study was to investigate the association between antidepressants use and the risk of Acute Myocardial Infarction (AMI).We conducted a nested case-control study using a primary care database over the period 2002-2015. From a

Antidepressant fluvoxamine reduces cerebral infarct volume and ameliorates sensorimotor dysfunction in experimental stroke.

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The sigma-1 receptor has been reported to be associated with diverse biological activities including cellular differentiation, neuroplasticity, neuroprotection, and cognitive functioning of the brain. Fluvoxamine, one of the currently known antidepressants, is a sigma-1 receptor agonist; its

Effects of antidepressant treatment following myocardial infarction.

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BACKGROUND Depression following myocardial infarction is associated with poor cardiac prognosis. It is unclear whether antidepressant treatment improves long-term depression status and cardiac prognosis. OBJECTIVE To evaluate the effects of antidepressant treatment compared with usual care in an

Antidepressant effect of lithium in patients with systemic lupus erythematosus and cerebral infarction, treated with corticosteroid.

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Two depressive patients with systemic lupus erythematosus (SLE) and cerebral infarction, while being treated with corticosteroid, are described. In both cases, lithium was remarkably effective and no severe side-effects occurred. These findings suggest that lithium can be a useful antidepressant

Effects of antidepressants on the intraventricular conduction and the incidence of arrhythmias induced by programmed ventricular stimulation in the dog heart after myocardial infarction.

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The effects of mianserin, a tetracyclic antidepressant, and adinazolam, a new triazolobenzodiazepine which has antidepressant activity, on intraventricular conduction and the incidence of arrhythmias induced by programmed ventricular stimulation were studied in the dog heart after myocardial

Comparative effects of antidepressants, amitriptyline, and maprotiline on intraventricular conduction, effective refractory period, and incidence of ventricular arrhythmias induced by programmed stimulation in dog hearts after myocardial infarction.

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The effects of amitriptyline and maprotiline, standard tricyclic and tetracyclic antidepressants, on intraventricular conduction, the effective refractory period (ERP), and the incidence of ventricular arrhythmias induced by programmed stimulation were studied and compared in dog hearts after

Antidepressant prescription in acute myocardial infarction is associated with increased mortality 1 year after discharge.

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To assess the impact of antidepressant (AD) prescription at discharge on 1-year outcome of patients presenting with acute myocardial infarction (AMI) in Switzerland.We used data from the AMIS Plus registry including patients admitted between March 2005 and

Antidepressant treatment with maprotiline in the management of emotional disturbances in patients with acute myocardial infarction: a controlled study.

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In coronary artery disease the patients usually manifest both anxiety and depression disturbances. A controlled clinical study was conducted to test the efficacy of a new antidepressant agent, maprotiline, in the early stages of acute myocardial infarction. The sample consisted of 126 patients,

Use of antidiabetic and antidepressant drugs is associated with increased risk of myocardial infarction: a nationwide register study.

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OBJECTIVE To explore the gender- and age-specific risk of developing a first myocardial infarction in people treated with antidiabetic and/or antidepressant drugs compared with people with no pharmaceutical treatment for diabetes or depression. METHODS A cohort of all Swedish residents aged 45-84

Effect of antidepressants and their relative affinity for the serotonin transporter on the risk of myocardial infarction.

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BACKGROUND Antidepressants, particularly selective serotonin reuptake inhibitors (SSRIs), attenuate platelet activation by depleting serotonin storage and may decrease risk of myocardial infarction (MI). These drugs differ in their affinity for the platelet serotonin transporter and therefore may

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