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Страница 1 от 73 полученные результаты
Inhibition of phytoalexin synthesis in arachidonic Acid-stressed potato tissue by inhibitors of lipoxygenase and cyanide-resistant respiration.
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Arachidonic acid-stressed potato tuber discs synthesized the phytoalexin rishitin. This synthesis was inhibited by salicylhydroxamic acid (SHAM), and to a lesser extent by tetraethylthiuram disulfide (disulfiram). Disulfiram was less effective apparently because it was inactivated in the tuber
Factors Influencing the Development of Cyanide-resistant Respiration in Potato Tissue.
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Ethylene, cyanide gas, and volatalized ethanol, acetaldehyde, and acetic acid were applied in a continuous flow to whole potato tubers. Freshly cut slices were obtained periodically during the treatment, and showed a progressive development of a cyanide-resistant respiration. The application of the
Reactive oxygen species induce cyanide-resistant respiration in potato infected by Erwinia carotovora subsp. Carotovora.
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Studies have shown that pathogenic bacteria infections induce the overproduction of reactive oxygen species (ROS) in plants. Cyanide-resistant respiration, an energy-dissipating pathway in plants, has also been induced by a pathogenic bacteria infection. However, it is unknown whether the induction
Cyanide sensitivity and induction of the microsomal oleoyl-CoA desaturase of potato tuber.
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1. The properties of oleoyl-CoA desaturase, induced in microsomal fractions by the 'ageing' treatment of potato tuber slices (aeration of slices for 3-18 h), were investigated to study the effect of cyanide on desaturation and cycloheximide on the induction of the desaturase. 2. The electrons needed
Cyanide-resistant Respiration of Sweet Potato Mitochondria.
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The oxidation of malate and succinate by sweet potato mitochondria (Ipomoea batatas [L.] Lam.) was blocked only partly by inhibitors of complexes III (2-heptyl-4-hydroxyquinoline-N-oxide) and IV (cyanide and azide). The respiration insensitive to inhibitors of complexes III and IV was inhibited by
Cyanide-resistant Respiration in Fresh and Aged Sweet Potato Slices.
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The respiration of fresh sweet potato (Ipomoea batatas) slices is resistant to, and often stimulated by, cyanide and antimycin A. m-Chlorobenzhydroxamic acid (CLAM), a selective inhibitor of the alternate path, inhibits respiration in the presence of cyanide and has a limited inhibitory effect in
Development of cyanide-resistant respiration in mitochondria from potato tubers treated with ethanol, acetaldehyde, and acetic acid.
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Treatment of intact potato (Solanum tuberosum L.) tubers with acetaldehyde, ethanol or acetic-acid vapors led to a respiratory upsurge which was further increased when the volatiles were applied in 100% O2. Mitochondria from tubers held in 100% O2 (O2 control) displayed a substrate state, state 3,
The mechanism of ethylene and cyanide action in triggering the rise in respiration in potato tubers.
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Ethylene and cyanide induce a sharp increase in respiration in potato tubers (Solanum tuberosum, var. Russet) attended by changes in the glycolytic intermediates which indicate that both gases enhance glycolysis. The level of sucrose also increases in response to both treatments. The data are taken
Inhibition of the Development of Induced Respiration and Cyanide-insensitive Respiration in Potato Tuber Slices by Cerulenin and Dimethylaminoethanol.
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The interdependence of the development of wound-induced respiration and membrane-related phospholipid biosynthesis in potato tuber (Solanum tuberosum var. Russet) slices was established by the use of agents which selectively affect lipid and phospholipid synthesis. Cerulenin, a specific inhibitor of
Relative Contribution of Cytochrome-mediated and Cyanide-resistant Electron Transport in Fresh and Aged Potato Slices.
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The respiration of fresh potato (Solanum tuberosum, var. Russet Burbank) slices is predominantly cyanide-sensitive whether in the presence or absence of uncoupler. By contrast, the wound-induced respiration which develops in thin slices with aging is cyanide-resistant, and in the presence of
Effects of Controlled Atmospheres on Production of Sesquiterpenoid Stress Metabolites by White Potato Tuber: Possible Involvement of Cyanide-resistant Respiration.
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Levels of katahdinone (solavetivone), lubimin, rishitin, and phytuberin, sesquiterpenoid stress metabolites of white potato (Solanum tuberosum), were monitored in tuber slices which were challenged with an extract of Phytophthora infestans and incubated under controlled atmospheres. A mixture of
Upsurge in respiration and peroxide formation in potato tubers as influenced by ethylene, propylene, and cyanide.
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A continuous application of ethylene (10 mul/l) and propylene (500 mul/l) to potato tubers (Solanum tuberosum L.) resulted in an upsurge of respiration and a concomitant rise in peroxides. When applied in 100% O(2), the effect of ethylene and propylene on respiration and peroxide formation was
Cyanide-resistant Respiration in Freshly Cut Potato Slices.
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Treating intact white potato (Solanum tuberosum L.) tuber with ethylene in air or O(2) made it possible to obtain freshly cut slices which exhibit cyanide-resistant respiration. The cyanide-resistant path requires induction in whole tubers. The data also indicate that high O(2) concentration is
The role of cyanide-resistant respiration in Solanum tuberosum L. against high light stress.
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The cyanide-resistant respiration that occurs in potato mitochondria is one of the important pathways for energy dissipation. It can be activated by glare. However, it is unclear what roles cyanide-resistant respiration play in the high-light stress of potatoes. This study designed a CRISPR vector
Reduction of water permeability in potato tuber slices by cyanide, ammonia, 2,4-dinitrophenol, and oligomycin and its reverse by adenosine 5'-triphosphate and cytidine 5'-triphosphate.
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Five millimolar KCN reduced water permeability in 1-millimeter thick slices of potato tuber (Solanum tuberosum L.). One-tenth millimolar ATP and CTP prevented or reversed the reduced permeability. UTP and GTP were not effective. Five millimolar ammonium carbonate or 0.1 millimolar 2,4-dinitrophenol
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