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anisodamine/ischemia
Odkaz sa uloží do schránky
Strana 1 od 18 výsledky
Anisodamine protects against neuronal death following cerebral ischemia in gerbils.
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OBJECTIVE
To study the effect of anisodamine on neuronal death and hydroxyl radical (OH.) production during forebrain ischemia-reperfusion in gerbils.
METHODS
The tested gerbils were divided into 3 groups, including sham-operated, control and anisodamine groups. In each group, there were 8 animals
The effects of anisodamine and dobutamine on gut mucosal blood flow during gut ischemia/ reperfusion.
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OBJECTIVE
To determine if anisodamine is able to augment mucosal perfusion during gut I/R ischemia-reperfusion.
METHODS
A jejunal sac was formed in Sprague Dawley rat. A Laser Doppler probe and a tonometer were inserted into the sac which was filled with saline. The superior mesenteric artery was
The effect of anisodamine on cerebral resuscitation of rats in acute cerebral ischemia from cardiac arrest.
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In order to investigate the mechanisms of acute cerebral ischemia, and to look for effective drugs on cerebral resuscitation, we made a model of acute complete global brain ischemia, reperfusion and resuscitation on rats according to Garavilla's method. Our results showed that the event of cerebral
[Experimental study on the protective effect of anisodamine on flaps with ischemia reperfusion injury].
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OBJECTIVE
To observe the effect of anisodamine (Ani) injection on the survival rate and histologic result of flaps with ischemia-reperfusion injury, so as to demonstrate the protective effect of Ani on the flap survival.
METHODS
A total of 48 healthy male Wistar rats were randomly divided into model
[Protective effects of bepridil in combination with anisodamine on myocardial ischemia and reperfusion injury in rats].
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The effects of bepridil (Bep) in combination with anisodamine (Ani) on myocardial ischemia and reperfusion injury induced by ligating the left coronary artery were studied in rats. Administration of a combination of Bep and Ani was found to decrease the incidence of arrhythmia induced by myocardial
Cardioprotective effects of anisodamine against myocardial ischemia/reperfusion injury through the inhibition of oxidative stress, inflammation and apoptosis.
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The aim of the present study was to investigate the cardioprotective effects of anisodamine against myocardial ischemia/reperfusion (I/R) injury and the molecular mechanisms involved. The present results demonstrated that anisodamine attenuated myocardial infarct sizes, decreased the levels of
Cardioprotective Effect of Anisodamine Against Myocardial Ischemia Injury and its Influence on Cardiomyocytes Apoptosis.
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Anisodamine is an ancient Chinese medicine derived from Tibet as a belladonna alkaloid, which is usually used for improvement of blood circulation in patients with organ phosphorus poisoning or shock. In this study, for the first time, we report its cardioprotective effects on preventing
Effects of anisodamine against myocardial ischemia-reperfused injuries and antilipid peroxidation.
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OBJECTIVE
Anisodamine (Ani), an alkaloid first isolated in China. To study the relationship between the protective effects of Ani on myocardial cells of reperfused injuries and the antilipid peroxidation.
METHODS
Coronary ligation for 15-min followed by 10 min reperfusion was performed in
Protective effect of anisodamine on reperfusion injury of skeletal muscles in rabbit.
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Anisodamine is an alkaloid isolated from a Chinese plant, which was subsequently synthesized. Its chemical structure is similar to atropine. It inhibits cholinergic nerve function, improves microcirculation, and was reported to have a protective effect on reperfusion injury in various organs. We
Inhibition of endoplasm reticulum stress by anisodamine protects against myocardial injury after cardiac arrest and resuscitation in rats.
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Anisodamine is a multi-functional bio-alkaloid with vascular activity. Our previous studies have revealed that anisodamine protects the heart from ischemia/reperfusion (I/R) injury induced by cardiac arrest (CA) and resuscitation. This study aimed to explore whether the protective effect of
Merit of anisodamine combined with opioid δ-receptor activation in the protection against myocardial injury during cardiopulmonary bypass.
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Myocardial ischemia/reperfusion (MIR) injury easily occurrs during cardiopulmonary bypass surgery in elderly patients. In an attempt to develop an effective strategy, we employed a pig model of MIR injury to investigate the maximum rate of change of left ventricular pressure, left ventricular
[Effects of 3 henbane drugs on acute forebrain ischemia and reperfusion injury in rats].
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After 30-min ischemia and 60-min reperfusions in rats by ligating bilateral vertebral and common carotid arteries, the brain calcium contents were increased from 171 +/- 6 micrograms in control group to 192 +/- 10 micrograms with abnormal EEG activities and ischemic injury in the brain tissues.
Cell protection mechanism of antishock action of anisodamine.
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Anisodamine was used as an antishock (chiefly septic) drug beginning in the early 1960s in China. Its underlining mechanism was believed to be due to its vasodilative action. But in normal animals it only produces slight vasodilation. Our work during the recent 15 years proved that anisodamine is
[Effect of hemorheological factors on coronary flow during myocardial ischemia].
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Intracoronary infusion of some drugs may induce superimposed coronary vasodilation upon endogenous vasodilation during myocardial ischemia, which was suggested as coronary vasodilator reserve. For an investigation of this phenomenon, 8 pigs were anesthetized, chest-opened, LAD (left anterior
[Protection of anisodamine on the mitochondrial injury induced by oxidative stress in swine with cardiac arrest].
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OBJECTIVE
To investigate the protection of anisodamine on cardiac ischemia/reperfusion (I/R) injury by oxidative stress by observing the changes in oxidation and antioxidant markers in plasma and myocardium, and the damage of cardiac mitochondria structure in pigs with cardiac arrest
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