antituberculosis/atrofia

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Clinical deterioration during antituberculosis treatment in Africa: incidence, causes and risk factors.

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BACKGROUND HIV-1 and Mycobacterium tuberculosis cause substantial morbidity and mortality. Despite the availability of antiretroviral and antituberculosis treatment in Africa, clinical deterioration during antituberculosis treatment remains a frequent reason for hospital admission. We therefore

Clinical spectrum of paradoxical deterioration during antituberculosis therapy in non-HIV-infected patients.

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Paradoxical deterioration during antituberculosis therapy, defined as the clinical or radiological worsening of pre-existing tuberculous lesions or the development of new lesions in a patient who initially improves, remains a diagnostic dilemma. Although different clinical presentations of

[Comparative study of hepatoprotective action of remaxol, reamberin and ademethionine in liver injury induced by antituberculosis drugs (experimental study)].

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The hepatoprotective activity of remaxol, reamberin and ademethionine was studied on a model of the liver injury induced by antituberculosis drugs. The study included 30 male uninbred albino rats. The following antituberculosis drugs were used: isoniazid (50 mg/kg) subcutaneously + rifampicin (250

Paradoxical response during anti-tuberculosis treatment in HIV-negative patients with pulmonary tuberculosis.

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BACKGROUND Transient worsening of tuberculosis (TB) symptoms and lesions following anti-tuberculosis treatment (paradoxical response [PR]), has been described in human immunodeficiency virus (HIV) infected patients who undergo anti-tuberculosis treatment. The frequency and clinical presentations for

Neurological manifestations and toxicities of the antituberculosis drugs. A review.

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The neurological manifestations and toxicities of 12 antituberculosis drugs [isoniazid, rifampicin (rifampin), ethambutol, p-aminosalicylic acid, pyrazinamide, streptomycin, kanamycin, ethionamide, cycloserine, capreomycin, viomycin and thiacetazone] are reviewed. Their effects upon the central

Seizures following Intoxication with a Common Antituberculosis Drug.

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Isoniazid is an antimycobacterial agent commonly prescribed in most tuberculosis chemotherapy regimens. Its side effects are widely known including mainly liver toxicity and peripheral neuropathy. The toxic effects of isoniazid are dose-related. Toxic doses are estimated at 35-40 mg/kg and fatal

[Paradoxical response during anti-tuberculosis treatment in immunocompetent patients].

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The exacerbation of clinical symptoms or radiological worsening of pre-existing tuberculous lesions or the development of new lesions defines paradoxical deterioration during antituberculosis therapy. This phenomenon is relatively frequent: it is observed between 6 and 30% of patients receiving

Exposure to first line anti-tuberculosis drugs in prepubertal age reduces the quality and functional competence of spermatozoa and oocytes in Swiss albino mice.

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Prepubertal Swiss albino mice of both sex were administered with first-line anti-tuberculosis drugs (ATDs) viz; rifampicin, isoniazid, pyrazinamide, streptomycin and ethambutol intraperitoneally, for 4 weeks. Two weeks after the completion of treatment, male mice were sacrificed to collect caudal

Paradoxical Reaction In The Form Of New Pulmonary Mass During Anti-Tuberculosis Treatment: A Case Series And Literature Review.

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Paradoxical reaction refers to deterioration of the original tuberculosis lesions or emergence of new infiltrative lesions during anti-tuberculosis treatment. The common manifestations of paradoxical reaction include new pleural effusion, cerebral tuberculosis and lymphadenitis.

Paradoxical Deterioration During Anti-Tuberculous Therapy in Non-HIV-Infected Patients with Pleural Tuberculosis: A Pragmatic Approach.

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We report a case of paradoxical deterioration. A male patient diagnosed with pleural tuberculosis, but who was not infected with human immunodeficiency virus (HIV), experienced clinical deterioration 3 weeks after the initiation of anti-tuberculous treatment. After other diagnoses were

Neurologic deterioration in a child undergoing treatment for tuberculosis meningitis.

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Clinical deterioration while receiving antituberculosis (anti-TB) therapy can be due to a number of etiologies, including drug resistance, disease progression despite effective therapy, or alternative diagnoses. We present the case of a 22-month-old girl diagnosed with TB meningitis 4 months prior

Risk factors for development of paradoxical response during antituberculosis therapy in HIV-negative patients.

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The risk factors for development of paradoxical response were studied in a cohort of 104 patients with culture-documented Mycobacterium tuberculosis infection. Paradoxical deterioration occurred in 16 (15.4%) patients (case group) during antituberculosis therapy, involving lungs and pleura (n=4),

Paradoxical reaction to antituberculosis therapy in a patient with lupus vulgaris.

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Patients receiving treatment for tuberculosis may experience an unexpected deterioration of their disease; this is known as a paradoxical reaction. We present the case of a 59-year-old man with lupus vulgaris who experienced a paradoxical deterioration of cutaneous lesions after starting

[Increase in size of lymph nodes or occurrence of new lymphadenopathy during antituberculosis chemotherapy: paradoxical response].

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The aim of this letter was to present four more cases of tuberculosis (TB) in whom paradoxical response developed. These presented cases had not been mentioned in the manuscript namely "Paradoxical radiologic progression despite appropriate antituberculous therapy" published in Mikrobiyol Bul 2012;

Paradoxical Reaction In The Form Of New Pulmonary Mass During Anti-Tuberculosis Treatment: A Case Series And Literature Review.

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