carbohydrate/hemorrhage

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The role of chronic alcohol intake in patients with spontaneous intracranial hemorrhage: a carbohydrate-deficient transferrin study.

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OBJECTIVE Chronic alcohol intake is considered to be a risk factor for spontaneous intracranial hemorrhage (SICH). However, there is a lack of objective data in this field. The aim was to assess its role in hemorrhagic stroke objectively and to evaluate its correlation with the elevation of

Pre-stress carbohydrate solution prevents fatal outcome after hemorrhage in 24-hour food-deprived rats.

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Twenty-four-hour food deprivation increases mortality after experimental hemorrhage. Survival after hemorrhage is closely related to the capacity of the animal to develop hyperglycemia. In this study, 24-h food-deprived rats were subjected to hemorrhage over a period of 75 min, standardized to reach

BIOCHEMICAL STUDIES ON SHOCK : I. THE METABOLISM OF AMINO ACIDS AND CARBOHYDRATE DURING HEMORRHAGIC SHOCK IN THE RAT.

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During and following the production of shock by hemorrhage in the normal, suprareno-demedullated, and suprarenalectomized rat, the following significant changes in amino acid and carbohydrate metabolism have been observed. 1. In the intact, suprareno-demedullated, and suprarenalectomized rat there

BIOCHEMICAL STUDIES ON SHOCK : II. THE ROLE OF THE PERIPHERAL TISSUES IN THE METABOLISM OF PROTEIN AND CARBOHYDRATE DURING HEMORRHAGIC SHOCK IN THE RAT.

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The changes in the blood levels of amino nitrogen, glucose, lactate, and pyruvate were compared in eviscerate (liverless) rats and eviscerate rats subjected to hemorrhage, in order to establish the rôle of the peripheral tissues in the blood changes during shock. It was found that:- 1. The blood

Metabolic networks in a porcine model of trauma and hemorrhagic shock demonstrate different control mechanism with carbohydrate pre-feed.

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BACKGROUND Treatment with oral carbohydrate prior to trauma and hemorrhage confers a survival benefit in small animal models. The impact of fed states on survival in traumatically injured humans is unknown. This work uses regulatory networks to examine the effect of carbohydrate pre-feeding on

[Changes in carbohydrate metabolism indices in patients with hemorrhagic fever associated with renal syndrome].

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Metabolism of carbohydrates was impaired in 80 patients with hemorrhagic fever accompanied by renal syndrome, in which activation of glycolysis occurred due to a decrease in aerobic oxidation and development of metabolic acidosis. Lactacidemia and activation of LDH in blood serum were considered as

[Isothiobarbamine prevention of disorders of carbohydrate and oxygen metabolism and of the development of brain edema in the early period after an intracerebral hemorrhage].

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In experiments on cats after injection of isothiobarbamine intravenously (50 mg/kg) at 30 min, intracerebral hemorrhage prevented activation of brain glucose utilization, depression of brain oxygen utilization, surplus lactate accumulation in brain, early development of brain edema and death of

[Dimethyl sulfoxide as a corrector of disorders of cerebral hemodynamics, oxygen and carbohydrate metabolism of the brain and lipid peroxidation in the early period after intracerebral hemorrhage].

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Intraperitoneal injection of DMSO (0.7 g/kg) in rats 1h after intracerebral hemorrhage prevented brain conjugated diens and fluorosciene products increase and limited cerebral ++anti-radical activity. In experiments on cats 1h after intracerebral hemorrhage intravenous infusion of DMSO (0.3 g/kg)

Effects of haemorrhage on myocardial consumption of oxygen and carbohydrate in fetal sheep in utero.

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A previous study has demonstrated that fetal myocardial blood flow decreased during haemorrhage. To assess the effects of a decrease in myocardial blood flow on fetal myocardial oxygenation and metabolism, we measured fetal myocardial consumption of oxygen, glucose, lactate, and pyruvate, at rest

A four-compartment metabolomics analysis of the liver, muscle, serum, and urine response to polytrauma with hemorrhagic shock following carbohydrate prefeed.

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OBJECTIVE Hemorrhagic shock accompanied by injury represents a major physiologic stress. Fasted animals are often used to study hemorrhagic shock (with injury). A fasted state is not guaranteed in the general human population. The objective of this study was to determine if fed animals would exhibit

Alterations in metabolite levels in carbohydrate and energy metabolism of rat in hemorrhagic shock and sepsis.

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For comparison of the extent of metabolite content alteration caused by etiologically different types of shock, septic peritonitis and hemorrhagic shock (mean arterial blood pressure at 40 mm Hg for 1 h or 2 h) were produced in rats. Contents of metabolites were determined in the liver and the

[Effect of glucagon on changes of carbohydrate metabolism in experimental hemorrhagic shock].

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The behaviour of venous blood glucose, lactate and pyruvate concentration was tested in experimental shock, caused by bleeding in rabbits. The animals of one group were retransfused after 1.5 hours hypotension of 40 mm Hg. The animals of another group were left with a total blood removal of 2% body

Imidazo[2,1-b]thiazole carbohydrate derivatives: Synthesis and antiviral activity against Junin virus, agent of Argentine hemorrhagic fever.

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Herein, we describe the syntheses of 3,5-disubstituted imidazo[2,1-b]thiazole. The cyclization step was performed in two different conditions by using either thermal or microwave heating. Comparing the results of both methodologies, we found that the microwave assistance is an improved alternative

[Characteristics of the change of carbohydrate function of the liver during clinical death and resuscitation under the conditions of combined trauma (burn and lethal blood loss)].

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[Regulation of carbohydrate metabolism in the process of restoration of the vital functions of the organism following lethal blood loss].

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