cerebral infarction/seizures

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Seizures and cerebral infarction in the full-term newborn.

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Cerebral arterial infarction is a more common cause of neonatal seizures than has been previously appreciated. In 50 full-term newborns with seizures studied, 7 had cerebral infarction which was the second most common definable cause of seizures. We describe these 7 full-term infants with

[Late onset seizures in cerebral infarction--clinical study of late onset seizures in cortical branch infarction].

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We retrospectively analyzed the clinical features, prognosis, CT scans and electroencephalograms in 527 patients with cerebral infarction in the Toranomon hospital between 1983 and 1990. CT scans revealed cortical involvement in 130 cases and subcortical infarcts in the territory of the perforating

Differential diagnosis between cerebral ischemia, focal seizures and limb shaking TIAs in moyamoya disease.

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We describe the clinical course of a young female Caucasian patient with bilateral moyamoya disease in whom we could diagnose the simultaneous occurrence of cerebral ischemia, TIAs, limb shaking TIAs and focal Jacksonian seizures. It is the second clinical communication in the literature elaborating

Seizure activity in the rat hippocampus, perirhinal and prefrontal cortex associated with transient global cerebral ischemia.

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Epileptiform EEG activity associated with ischemia can contribute to early damage of hippocampal neurons, and seizure activity may also lead to dysfunction in extrahippocampal regions. In this study, seizure activity associated with the four-vessel occlusion model of cerebral ischemia was monitored

Neonatal cerebral infarction: an under recognised/unreconised cause of neonatal seizures?

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Neonatal cerebral infarction or neonatal stroke is a well recognised cause of neonatal seizures. The awareness of its existence among healthcare workers is low and it is not even mentioned as a cause of neonatal seizures in most books. Many of the affected neonates are well, without any evidence of

Cerebral infarction as multifocal clonic seizures in a term neonate.

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BACKGROUND The incidence of neonatal stroke in full-term infants has been cautiously estimated as 1:10,000, but infants can initially have few symptoms, and the condition has the potential for underdiagnosis. Follow-up studies of known full-term neonatal stroke victims beyond 3 years of age indicate

Epileptic seizures following ischaemic cerebral infarction. Clinical picture, CT findings and prognosis.

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Clinical, CT and EEG findings of 100 consecutive patients suffering from epileptic seizures following hemispheric cerebral infarction were evaluated retrospectively. All patients were followed up for an average of 49 months. Twelve patients suffered from cardiogenic brain embolism, 20 had an

Correlation of tibial nerve SEPs with the development of seizures in patients with supratentorial cerebral infarcts.

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In 40 patients with supratentorial non-haemorrhagic cerebral infarct, the findings in the tibial nerve SEPs recorded during the acute stage correlated significantly with the development of seizures during a 1 year follow-up period. Abnormality in the side-to-side difference of the P40-N48 amplitude

Recurrent seizures following focal motor status epilepticus in a patient with non-ketotic hyperglycemia and acute cerebral infarction.

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Focal motor status epilepticus (FMSE) is often associated non-ketotic hyperglycemia (NKH). There are no previous reports describing FMSE with NKH that was accompanied by an acute cerebral infarction and its long term follow-up result. We describe the case of a patient having focal motor status

Late epileptic seizures after cerebral infarction.

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OBJECTIVE To determine the profile of late epileptic seizures following cerebral infarcts and the predictive clinical and radiological factors associated with their development. METHODS We compared 86 patients who developed late seizures after cerebral infarction with 285 similar patients who did

Cerebral infarcts and seizures in the neonate.

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A retrospective analysis was performed on 54 infants who suffered perinatal hypoxic-ischemic insults and came to autopsy, in an attempt to assess the association of cerebral infarcts with seizures. Fifty infants had several types of cerebrovascular lesions, including intraventricular hemorrhage (32

Effect of inherent epileptic seizures on brain injury after transient cerebral ischemia in Mongolian gerbils.

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Subthreshold excitotoxic stimuli such as brief cerebral ischemia or chemically induced seizures modulate brain injury resulting from subsequent transient ischemia. Depending on the delay between the two insults, either tolerance or cumulative damage will develop. We were interested whether

Development of susceptibility to audiogenic seizures following cardiac arrest cerebral ischemia in rats.

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Susceptibility to audiogenic seizures (AGS) was investigated in Sprague-Dawley rats subjected to cardiac arrest cerebral ischemia (CACI), produced by compression of the major cardiac vessels. The onset of AGS was regularly observed 1 day after CACI of > 5 min duration. The duration of postischemic

Glycerol as an indicator of lipid degradation in bicuculline-induced seizures and experimental cerebral ischemia.

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Glycerol, the end product of phospholipid degradation, was measured in cat brains under pathophysiological conditions known to cause activation of lipolysis, namely, bicuculline-induced seizures, permanent focal cerebral ischemia (2 hr of middle cerebral artery occlusion), and global cerebral

Delayed ischemic electrocortical suppression during rapid repeated cerebral ischemia and kainate-induced seizures in rat.

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Global cerebral ischemia induces, within seconds, suppression of spontaneous electrocortical activity, partly due to alterations in synaptic transmission. In vitro studies have found that repeated brief hypoxic episodes prolong the persistence of synaptic transmission due to weakened adenosine

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