thymidine/atrofia

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Identification of the thymidine kinase gene of feline herpesvirus: use of degenerate oligonucleotides in the polymerase chain reaction to isolate herpesvirus gene homologs.

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Feline herpesvirus 1 (FHV) is the causative agent of viral rhinotracheitis in cats. Current vaccination programs employing attenuated live and killed FHV vaccines have been effective in reducing the incidence of this disease. As an initial step in the development of recombinant FHVs for use in the

The fine structure of pulse labeled (3-H-thymidine cells) in degenerating rat optic nerve.

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The ultrastructure of pulse labeled (3-H-thymidine) cells in rat optic nerve undergoing Wallerian degeneration is described. The study was limited to the first ten days after enucleation since cell proliferation during this interval is greater than in normal optic nerve (Skoff and Vaughn, '71).

Transneuronal degeneration of thalamic neurons following deafferentation: quantitative studies using [3H]thymidine autoradiography.

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Transneuronal degeneration of thalamic neurons following partial deafferentation was studied using [3H]thymidine autoradiography. Timed-pregnant female Sprague-Dawley rats received systemic injections of [3H]thymidine on embryonic day (E) 13, 14 and/or 15. On the day of birth, pups were anesthetized

Rapid appearance of labeled degenerating cells in the dorsal root ganglia after exposure of chick embryos to tritiated thymidine.

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The interval between [3H]thymidine delivery and onset of cellular degeneration in 5.5 day embryonic chick brachial dorsal root ganglia was examined autoradiographically. Of the degenerating cells, 14% were labeled by 2 h after [3H]thymidine delivery. This percentage increased for 24 h. Wing bud

Autoradiographic labelling of pancreatic acinar cells with thymidine-H3 during degeneration and regeneration.

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The uptake of thymidine-H(3) in the acinar nuclei of the rat pancreas was studied by autoradiography after segmental excision of the pancreas, ligation of a segmental pancreatic duct, or ligation of the artery to the splenic segment of the pancreas. No sustained increase of nuclear labelling

[Studies on nutritional management in operation. 2. A study on the influence of 19-nor-adrostenolone phenylpropionate administration on the suprarenal gland. 2. Concerning 3H-thymidine uptake and the chemical changes in this suprarenal cortex tissue in the presence of degeneration, atrophy, regeneration and proliferation].

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Failure of short-term luminal IGF-I to protect against atrophy in a model of fetal esophageal atresia.

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Short-term luminal infusion in utero (3 days) of insulin-like growth factor I (IGF-I) failed to protect the fetal small intestine against atrophy induced by ablation of swallowing. Human recombinant IGF-1 (or vehicle) was infused into the duodenum of fetal sheep at 125 days' gestation for 3 days

Dynamics of epithelial cells in the corpus of the mouse stomach. IV. Bidirectional migration of parietal cells ending in their gradual degeneration and loss.

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The life story of parietal cells has been investigated in the corpus of the mouse stomach using electron microscopy and 3H-thymidine radioautography. Parietal cells are scattered in the four regions of the unit. On the average 3.6 cells are in the pit, 6.2 in the isthmus, 5.6 in the neck, and 10.6

Degeneration of myelinated efferent fibers prompts mitosis in Remak Schwann cells of uninjured C-fiber afferents.

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The factors inducing normally innervated Schwann cells in peripheral nerve to divide are poorly understood. Transection of the fourth and fifth lumbar ventral roots (L4/5 ventral rhizotomy) of the rat is highly selective, sparing unmyelinated axons and myelinated sensory axons; Wallerian

Mechanism of androgen-induced thymic atrophy in the wall lizard, Hemidactylus flaviviridis: an in vitro study.

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The present in vitro study demonstrates the effect of androgen on thymocyte apoptosis leading to thymic atrophy in the wall lizard, Hemidactylus flaviviridis. Thymocytes collected from castrated lizards were incubated with varying concentrations of dihydrotestosterone (DHT) to observe its effect on

Correlation between serum high density lipoprotein content and liver function during experimental hepatic degeneration and regeneration.

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Intravenous injection of praseodymium nitrate into female Wistar rats results in liver damage. The aim of this study is to investigate the quality of serum high density lipoprotein content as an index for the severity and time course of liver damage and regeneration following the administration of

Investigations on the turnover of adrenocortical mitochondria. VI. An autoradiographic study of the effect of ACTH on the radioactivity decay in the mitochondrial compartment from the zona fasciculata of 3H-thymidine injected rats.

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The radioactivity decay of the mitochondrial compartment from the zona fasciculata of the adrenal cortex of 3H-thymidine-injected rats was followed by high resolution autoradiography. The number of days in which the radioactivity of the mitochondrial compartment was reduced to a half was calculated

The role of apoptosis in the development of renal cortical tubular atrophy associated with healed experimental renal papillary necrosis.

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An animal model of chronic analgesic nephropathy, in which renal papillary necrosis was induced by the administration of a single injection of bromoethylamine 2-hydrobromide in male Sprague-Dawley rats, was used to investigate the pathogenesis of the atrophy of tubules that leads to cortical atrophy

Quantification of the mononuclear phagocyte response to Wallerian degeneration of the optic nerve.

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We investigated the numbers, origin and phenotype of mononuclear phagocytes (macrophages/microglia) responding to Wallerian degeneration of the mouse optic nerve in order to compare it with the response to Wallerian degeneration in the PNS, already described. We found macrophage/microglial numbers

Cyclin expression in the atrophying and proliferating lobes of the liver after portal vein branch ligation and hepatectomy in rats.

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BACKGROUND Portal vein branch ligation causes atrophy of the portal vein ligated lobes (PVL) and proliferation of the nonligated lobes (PVNL) of the liver. However, the mechanisms underlying atrophy of the PVL and proliferation of PVNL after portal vein branch ligation have not been clarified except

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