toxicodendron rhomboideum/erythema

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Erythema multiforme associated with contact dermatitis to poison ivy: three cases and a review of the literature.

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Erythema multiforme (EM) is a hypersensitivity reaction that occurs mainly after exposure to certain medications or in the setting of infection, most commonly that due to herpes simplex virus. Rare cases of EM have been reported after allergic contact dermatitis due to various substances. There has

Erythema multiforme associated with Rhus contact dermatitis.

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A case of erythema multiforme and Rhus contact dermatitis is presented herein. Due to the association of nephritis and other complications with Rhus poison oak, we recommend that patients with contact dermatitis due to poison ivy who incur secondary erythema multiforme be hospitalized and closely

Poison ivy: an underreported cause of erythema multiforme.

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The relationship between herpes simplex virus (HSV) and erythema multiforme (EM) has been well described. Many authors contend that EM (excluding Stevens-Johnson syndrome and toxic epidermal necrolysis) occurs almost exclusively as a response to HSV infection. During the past year, however, we have

Vesicular Contact Reaction May Progress into Erythema Multiforme.

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Dear Editor, Erythema multiforme is considered an acute skin condition, characterized by a self-limiting and sometimes recurrent course. It is regarded as a type IV hypersensitivity reaction associated with certain infections, medications, and other various triggers. Allergic contact dermatitis is

Plant dermatitis. Possible culprits go far beyond poison ivy.

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Given the variety of existing plant species in the environment, it is remarkable that people have adjusted as well as they have to the many plants that can cause uncomfortable skin reactions. With a basic understanding of the types of reaction and the common plants that cause each type, physicians

Preservation of allergic contact dermatitis to poison ivy (urushiol) in late HIV disease. The implications and relevance to immunotherapy with contact allergens.

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BACKGROUND Delayed hypersensitivity reactions (DTH) are lost with progression of HIV disease. This loss of DTH commonly occurs before the onset of opportunistic infections and is an independent predictor of disease progression. OBJECTIVE We wanted to determine whether patients in late HIV disease

Modulation of leucocyte adhesion molecules, a T-cell chemotaxin (IL-8) and a regulatory cytokine (TNF-alpha) in allergic contact dermatitis (rhus dermatitis).

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To understand the molecular events which are important in leucocyte trafficking in cutaneous inflammation, poison ivy/oak extract was applied topically to the skin, and the simultaneous assessment of a variety of clinical and immunopathological parameters performed. The clinical response of subjects

In vivo documentation of cutaneous inflammation using spectral imaging.

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Typical manifestations of cutaneous inflammation include erythema and edema. While erythema is the result of capillary dilation and local increase of oxygenated hemoglobin concentration, edema is characterized by an increase in extracellular fluid in the dermis, leading to local tissue swelling.

The summer penile syndrome: seasonal acute hypersensitivity reaction caused by chigger bites on the penis.

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OBJECTIVE To describe the seasonal acute hypersensitivity reaction of the penis due to chigger bites, known as the summer penile syndrome. METHODS A consecutive series of patients. METHODS The emergency department of an urban academic children's hospital in the midwestern United States. METHODS Male

[Contact allergy caused by poison ivy (Toxicodendron spp].

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Within 3 days two female patients presented with an eruption featuring erythema, edema, vesicles and bullae. We suspected poison ivy allergy which was subsequently proven by history and positive patch tests. In Germany the risk of specific hypersensitivity to Toxicodendron species is low, since the

Diagnosis and management of contact dermatitis.

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Contact dermatitis is a common inflammatory skin condition characterized by erythematous and pruritic skin lesions that occur after contact with a foreign substance. There are two forms of contact dermatitis: irritant and allergic. Irritant contact dermatitis is caused by the non-immune-modulated

Prevention of poison ivy and poison oak allergic contact dermatitis by quaternium-18 bentonite.

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BACKGROUND Poison ivy and poison oak are the most common causes of allergic contact dermatitis in North America. OBJECTIVE We investigated whether a new topical lotion containing 5% quaternium-18 bentonite prevents experimentally induced poison ivy and poison oak allergic contact

Keratinocyte intercellular adhesion molecule-1 (ICAM-1) expression precedes dermal T lymphocytic infiltration in allergic contact dermatitis (Rhus dermatitis).

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The ability of small molecules such as urushiol, present as a wax on the poison ivy leaf surface, to cause allergic contact dermatitis (rhus dermatitis) has fascinated immunologists for decades. Current dogma suggests that these epicutaneously applied catechol-containing molecules serve as haptens

Toxicodendron antigen patch test. Degrees of inflammation observed after various time intervals.

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Allergic contact dermatitis was elicited with Toxicodendron antigen and the patch test site examined at various time intervals up to one week. The degree of inflammation was rather constant during the observation period. The mean erythema score at 168 hours was not significantly different from the

Clinical and immunologic features of systemic contact dermatitis from ingestion of Rhus (Toxicodendron).

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Oral or parenteral exposure to certain contact allergens may elicit an eczematous skin reaction in sensitized individuals. This phenomenon has been called systemic contact dermatitis (SCD) and is relatively rare when compared with classical contact dermatitis. We reviewed and analysed the clinical

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