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glutamic acid decarboxylase/driska

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ČlankiKliničnih preskušanjPatenti
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[Isaacs' syndrome, stiff person syndrome and Satoyoshi disease: pathomechanisms and treatment].

Samo registrirani uporabniki lahko prevajajo članke
Prijava / prijava
Neurological disorders with characteristic clinical manifestations of painful muscle cramp and stiffness are not infrequent. The immune-mediated mechanism with specific antibodies among these diseases is particularly important for treatment. Isaacs' syndrome (acquired neuromyotonia) is an
The ability of enteropathogenic Escherichia coli (EPEC) to express virulence factor genes and develop attaching and effacing (AE) lesions is inhibited in acidic environmental conditions. This inhibition is due to the activation of transcription factor GadX, which upregulates expression of

A case of fulminant type 1 diabetes mellitus accompanied by myocarditis.

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Prijava / prijava
This report presents the case of a 47-year-old female patient with fulminant type 1 diabetes mellitus and myocarditis. Following a high fever, nausea, vomiting and diarrhea, diabetic ketoacidosis occurred and she was transferred to the hospital. The plasma glucose level was 63.6 mmol/L and HbA1c was

A novel role of intestine epithelial GABAergic signaling in regulating intestinal fluid secretion.

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Prijava / prijava
γ-Aminobutyric acid (GABA) is the primary inhibitory neurotransmitter in the central nervous system, and it is produced via the enzymatic activity of glutamic acid decarboxylase (GAD). GABA generates fast biological signaling through type A receptors (GABA(A)R), an anionic channel. Intriguingly,
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