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herpes genitalis/otok
Веза се чува у привремену меморију
Страна 1 од 413 резултати
Non-immune hydrops fetalis caused by herpes simplex virus type 2 in the setting of recurrent maternal infection.
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We report a case of non-immune hydrops fetalis (NIHF) caused by herpes simplex virus type 2 (HSV-2) in an infant whose mother had recurrent HSV-2 infection. In spite of prematurity, severe disseminated infection and hydrops, the infant survived and was neurologically intact. HSV-2-induced NIHF is
Detection of human herpes virus 6 DNA in fetal hydrops.
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Human herpes virus 6 (HHV6) DNA was detected in two of eight fetuses with hydrops and none of ten non-hydropic dead fetuses. Both cases with HHV6 DNA had chromosomal abnormalities. Positive results were confirmed with a second PCR specific for an alternate region of the HHV6 genome. Restriction
Non-immune hydrops associated with congenital herpes simplex infection.
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Non-immune hydrops fetalis is a rare pregnancy complication, which can be caused by a myriad of conditions. Etiologies include metabolic or chromosomal disorders, fetal structural anomalies, and fetal infections. We present a case of non-immune hydrops caused by an intra-uterine infection with Type
Edema Bullae Mimicking Disseminated Herpes Zoster.
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Edema bullae typically forms at the site of skin swelling during acute states of volume overload, most commonly during renal or cardiac failure. Herpes zoster is a reactivation of latent varicella zoster virus that typically presents as painful vesicles in a dermatomal distribution. In
Descemet Membrane Endothelial Keratoplasty in Irreversible Corneal Edema Due to Herpes Simplex Virus Endotheliitis.
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Therapeutic response of herpes simplex virus-induced corneal edema to trifluridine in combination with immunosuppressive agents.
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Herpetic stromal disease often is treated with combinations of antiviral agents and corticosteroids. The addition of steroids to the antiviral treatment regimen frequently increases the efficacy of therapy in patients; however, many complications may arise as a result of corticosteroid therapy.
Acute hemorrhagic edema of infancy associated with herpes simplex type 1 stomatitis.
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Acute hemorrhagic edema of infancy is a benign leukocytoclastic vasculitis occurring in children younger than 2 years. The etiology is unknown. Viral or bacterial infections, immunizations, and the use of several medications, mainly antibiotics, may be involved in the pathogenesis. We report the
The development of corneal edema in herpes simplex virus type 1-infected rabbits following termination of therapy for corneal stromal disease.
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One complication of combined antiviral/corticosteroid therapy for herpetic stromal disease in patients is rebound of disease upon termination of therapy. To develop a model of steroid rebound, rabbits were injected intrastromally with 10(3) pfu of HSV-1 (RE strain). Therapy with 1%
EFFECT OF LOCAL EDEMA AND INFLAMMATION IN THE SKIN OF THE MOUSE ON THE PROGRESSION OF HERPES VIRUS.
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Nonimmune hydrops fetalis due to herpes simplex virus type 1.
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Hydrops fetalis: an unusual presentation of intrauterine herpes simplex virus infection.
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[Recurrent burning pain, erythema, cutaneous edema and hyperthermia of both lower legs after herpes zoster thoracalis].
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[Early diagnosis of herpes simplex virus encephalitis by single photon emission computed tomography (SPECT) in patients with normal MRI].
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Since treatment of herpes simplex virus encephalitis (HSVE) is most effective when started early, a sensitive and specific method for early diagnosis would be of great benefit. MRI and CT are commonly used for this purpose. In this study, we presented two patients who had serologically confirmed
Glioblastoma mimicking herpes simplex encephalitis.
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We report a case of 70-year-old man with glioblastoma presenting as acute encephalitic illness. The patient exhibited sudden onset of cognitive impairment and headache for 2 days. Initial brain MRI showed left temporal lobe hyperintensity, and cerebrospinal fluid cytology revealed a mild
Good prognosis for psychomotor development in survivors with nonimmune hydrops fetalis.
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We examined the psychomotor development of 33 of 61 surviving children, from a series of 107 consecutive live-born cases with nonimmune hydrops fetalis. The majority had a normal outcome. Three had a (simultaneous) serious underlying disease (e.g. fetal herpes infection) and had either severe
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