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Страна 1 од 79 резултати
Ketogenic diet prevents seizure and reduces myoclonic jerks in rats with cardiac arrest-induced cerebral hypoxia.
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Although the mechanism underlying the anti-epileptic effects of a ketogenic diet (KD) is not known, KD is reported to be an effective treatment for intractable epilepsy, in particular among children. Here, we evaluated whether a KD can reduce posthypoxic seizure and myoclonic jerks in a rat model of
Amiloride but not memantine reduces neurodegeneration, seizures and myoclonic jerks in rats with cardiac arrest-induced global cerebral hypoxia and reperfusion.
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It has been reported that both activation of N-methyl-D-aspartate receptors and acid-sensing ion channels during cerebral ischemic insult contributed to brain injury. But which of these two molecular targets plays a more pivotal role in hypoxia-induced brain injury during ischemia is not known. In
Thalamic deep brain stimulation for the treatment of action myoclonus caused by perinatal anoxia.
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BACKGROUND
Perinatal anoxia rarely causes myoclonus as the main neurologic abnormality. The exact neuronal mechanism underlying myoclonus induced by perinatal anoxia remains unknown. Some studies have indicated that the development of involuntary movements may be related to the maturation of the
Action myoclonus after cerebral anoxia.
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Intention or kinesogenic myoclonus is uniquely one of many different movement disorders that if recognized and treated, early or late, responds to treatment. Posthypoxic intention myoclonus appears to be a syndrome associated with a variety of interacting biochemical mechanisms.
Selective stimulus-sensitive myoclonus in acute cerebral anoxia. A case report.
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We report a 69-year-old patient, a chronic diabetic, who had a cardiac arrest after myocardial infarction. In the state of acute anoxic coma, massive myoclonic jerks occurred, and it was found that myoclonus could be elicited by light touch of the right trigeminal area (but of no other body region).
Action myoclonus following acute cerebral anoxia.
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Action myoclonus secondary to posthypoxic encephalopathy is being seen increasingly with improved resuscitation techniques. A case report describes 5 specific physical and occupational therapeutic techniques for achieving independence in ambulation, transfers and self-care: (1) analysis and
[Prognostic value of certain forms of myoclonus after cerebral anoxia E.E. G. findings].
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[Myocloni following cerebral hypoxia (action-intention myoclonus)].
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Action myoclonus after cerebral hypoxia: treatment with clonazepam.
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NMDA receptor-mediated excitotoxicity contributes to the cerebral hypoxic injury of a rat model of posthypoxic myoclonus.
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Cardiac arrest-induced cerebral hypoxic injury could induce posthypoxic movement disorders. Here we investigated the effects of memantine, an NMDA receptor channel blocker, on the neurodegeneration occurred in an established rat model of posthypoxic myoclonus. We found that administration of
Postanoxic symptomatic oscillatory myoclonus.
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Two young patients with a history of birth anoxia and cerebral palsy developed paroxysmal irregularly oscillating muscle jerks at rest and during voluntary movement. Electrophysiologic studies revealed short bursts of alternating activity at 4 to 6 Hz in antagonist muscles. The absence of
Carotid brainstem reflex myoclonus after hypoxic brain damage.
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A patient comatose after acute anoxia developed bilaterally synchronous, periodic myoclonic jerks most prominently in the bilateral upper limbs. Although the myoclonus seemed to occur spontaneously, electrophysiological studies showed that the myoclonic jerks correlated in timing and size with
[Prognostic significance of myoclonias in aerospace altitude studies and others with acute hypoxia-accompanied status].
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Motor phenomena - mostly in the form of myoclonus in the widest sense - are not rare in aeromedical studies conducted at flight altitudes and involving severe and acute hypoxia. At the same time medium to severe general changes can be observed in the EEG. When oxygen is supplied the motor phenomena
Serial MRI findings in brain anoxia leading to Lance-Adams syndrome: a case report.
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Although in literature almost 150 patients with Lance-Adams Syndrome (LAS) have been reported, neuroradiological evaluations were often performed in late stages and there is no serial study evaluating LAS from early stages. We herein report a serial neuroimaging study demonstrating early and
Impact of transient acute hypoxia on the developing mouse EEG.
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Hypoxemic events are common in sick preterm and term infants and represent the most common cause of seizures in the newborn period. Neonatal seizures often lack clinical correlates and are only recognized by electroencephalogram (EEG). The mechanisms leading from a hypoxic/ischemic insult to acute
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