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Cystoid macular edema induced by low doses of nicotinic Acid.

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Cystoid macular edema (CME) is a condition that involves the macula, causing painless vision loss. In this paper, we report a case of niacin-induced bilateral cystoid macular edema (CME) in a middle-age woman taking low dose of niacin (18 mg of nicotinic acid). Optical coherence tomography (OCT)

[Use of nicotinic acid and midocalm for correcting blood coagulation in patients with radiation edema of the extremities].

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A study was made of the functional activity of platelets, plasmatic coagulation and fibrinolysis in patients with late radiation disorders of the blood and lymph flow during treatment with vasodilative drugs: nicotinic acid and midocalm. These drugs tend to decrease the functional activity of

[THE ALTERATION OF EXPERIMENTAL TRAUMATIC BRAIN EDEMA IN RATS WITH A NICOTINIC ACID-THEOPHYLLINE COMPOUND].

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[Nicotinic acid ester reaction and histamine-induced edema influence of air currents of various temperatures].

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[Experimental brain edema caused by 6-aminonicotinamide--an antimetabolite of nicotinic acid].

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Macular edema associated with nicotinic acid (niacin)

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[The duration of the action of a single dose of aspirin, kurantil and nicotinic acid during pregnancy complicated by EPH gestosis].

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Disorders in the system of blood aggregation state control (BASC) reflects the severity of OPH-gestosis (edema, proteinuria, hypertension). The duration of the therapeutic effect of the applied antithrombotics has not been studied essentially. The parameters and duration of the effective responses

Nicotinic acid treatment for Paralepistopsis acromelalga intoxication: assessment using magnetic resonance imaging.

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BACKGROUND Paralepistopsis acromelalga, formerly known as Clitocybe acromelalga, is a rare poisonous mushroom. The mycotoxins in this mushroom cause symptoms resembling those of erythromelalgia; however, its pathogenesis remains unclear. In this report, a patient who received nicotinic acid

Potential role for epidermal Langerhans cells in nicotinic acid-induced vasodilatation in the mouse.

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OBJECTIVE Our objective is to study the role of cutaneous Langerhans cells on a mouse model of nicotinic acid-induced vasodilatation. METHODS Nicotinic acid-induced vasodilatation was studied in the mouse ear by laser Doppler flowmetry prior to and at intervals after Langerhans cells depletion by

[Cystoid macular edema].

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Cystoid macular edema is a known side effect of different systemic and local medications. Nicotinic acid used as a hypolipemiant agent can cause cystoid macular edema. Local adrenergic antiglaucomatous drugs as well as prostaglandin analogs can induce cystoid macular edema especially if other risk

Nicotinic acid induces antinociceptive and anti-inflammatory effects in different experimental models.

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Although in vitro studies have shown that nicotinic acid inhibits some aspects of the inflammatory response, a reduced number of in vivo studies have investigated this activity. To the best of our knowledge, the effects induced by nicotinic acid in models of nociceptive and inflammatory pain are not

NUTRITIONAL CYTOPENIA IN MONKEYS RECEIVING THE GOLDBERGER DIET.

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Experiments are reported upon young rhesus monkeys which were given a diet essentially the same as the Goldberger black tongue-producing diet, supplemented in various ways. Those receiving the unsupplemented diet developed the syndrome characterized by leucopenia, anemia, gingivitis, diarrhea, and

Acanthosis nigricans--decreased extracellular matrix viscosity: cancer, obesity, diabetes, corticosteroids, somatotrophin.

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Acanthosis nigricans is a reaction pattern to over a dozen different causes. The skin, most classic in the axilla, is dark, soft, velvet-like with fine folding and papillae. The mechanism of this skin change is decreased viscosity of extracellular matrix (ECM) combined with mechanical extrusion of

Pellagra--increased viscosity of extracellular matrix.

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Pellagra is the result of increased viscosity of extracellular matrix (ECM). The skin changes in pellagra localize at sites of sun exposure or trauma because the mediators which are released cannot diffuse from the area. Edema is reported as the first clinical and microscopic change. Changes in the

Abnormal cutaneous reactions in dermatitis herpetiformis.

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In 94% of the patients with dermatitis herpetiformis, a locally applied ointment with an ester of nicotinic acid (Trafuril) induced an abnormal reaction with erythema, edema, papules, and often vesicles. The appearance of the reaction to Trafuril is similar to DH lesions. It differs markedly from

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