adenoma/fetma

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Obesity is common at diagnosis of childhood pituitary adenoma, and may persist following successful treatment.

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There is a paucity of data describing long-term outcomes of paediatric patients with pituitary adenoma. In this report we describe clinical features, treatment and outcomes of a paediatric cohort.Retrospective cohort study.24

Weight Change and Obesity Are Associated with a Risk of Adenoma Recurrence.

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Obesity is reportedly a risk factor for colorectal adenoma. However, the influence of weight change and obesity on colorectal adenoma recurrence is unclear. To investigate whether weight change and obesity are associated with recurrence and development of colorectal adenoma. Of 62,171 asymptomatic

A Hepatocellular Adenoma in a Diet-induced Obese Mouse.

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A hepatic nodule was noted in a C57BL/6J mouse with diet-induced obesity at 53 weeks of age. Macroscopically, a protruding yellowish white nodule was observed on the visceral surface of the left lateral lobe. Light microscopy demonstrated clear demarcation from the compressed adjacent parenchyma,

The relation of smoking, alcohol use and obesity to risk of sigmoid colon and rectal adenomas.

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We conducted a case-control study, using 429 cases with histologically confirmed sigmoid adenoma, 75 cases with rectal adenoma, and 3101 controls showing normal colonoscopy at least up to 60 cm from the anus. The subjects were male Self-Defense Forces personnel aged 48-56 who received a retirement

Silent pituitary adenoma and metabolic disorders: obesity, abnormal glucose tolerance, hypertension and dyslipidemia

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A silent pituitary adenoma (SPA) is characterized by the expression of pituitary hormones, detected by immunohistochemical staining, in the absence of clinical signs or symptoms of hormonal excess. Compared with functional pituitary adenomas, little is known regarding the involvement of SPAs in

Role of obesity in a randomized placebo-controlled trial of difluoromethylornithine (DFMO) + sulindac for the prevention of sporadic colorectal adenomas.

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BACKGROUND Chemoprevention with the polyamine-inhibitory regimen difluoromethylornithine (DFMO) + sulindac markedly reduces risk of recurrent adenoma in colorectal adenoma patients. Obesity is associated with risk of colorectal adenoma and colorectal cancer. This study investigates how obesity

Regression of hepatocellular adenoma after bariatric surgery in severe obese patients.

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OBJECTIVE Obesity and metabolic syndrome are increasingly recognized as risk factors for hepatocellular adenomas (HCA). There is still sparse evidence whether weight loss or bariatric surgery could induce HCA regression in these patients. In this brief report we describe the effect of weight loss on

Hepatocellular adenoma subtypes: the impact of overweight and obesity.

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BACKGROUND Hepatocellular adenomas (HCA) are rare benign tumours occurring mainly in women using oral contraceptives. With the appearance of a generation of new oral contraceptives, the number of HCA cases was anticipated to decrease, but in fact this was not observed in our practice. The influence

Case of resected multiple hepatocellular adenomas in a young man with severe obesity.

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Hepatocellular adenoma (HCA) is a rare liver tumor that has the potential for rupture and malignant transformation. Here, we report a case of multiple hepatocellular adenomas (HCAs) that were treated by surgical resection.

CASE PRESENTATION

Do factors related to endogenous and exogenous estrogens modify the relationship between obesity and risk of colorectal adenomas in women?

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Obesity has consistently been associated with increased colorectal cancer risk in men, but not in women. In the absence of postmenopausal hormone use (PMH), adipose-derived estrogen is the primary determinant of circulating estrogen levels in postmenopausal women, perhaps ameliorating the mitogenic

Physical activity, obesity, and risk of colorectal adenoma in women (United States).

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The relationship between physical inactivity, body mass index (BMI) (wt[kg]/ht[m]2), and pattern of adipose distribution with risk of colorectal adenomas (precursors of cancer) was examined in 13,057 female nurses in the United States, 40 to 65 years of age in 1986, who had an endoscopy between 1986

Two familial giant pituitary adenomas associated with overweight: clinical, morphological and genetic features.

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OBJECTIVE Pituitary adenomas are usually sporadic, although rare familial cases have been described. Here we report two first degree female cousins with giant pituitary adenoma and overweight. Both presented with secondary amenorrhoea, occasional headache and weight gain. METHODS In both patients

Obesity, weight change, and risk of adenoma recurrence: a prospective trial.

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OBJECTIVE Obesity is a risk factor for colorectal neoplasia. Lifestyle modifications, including weight loss, have been advocated to reduce the risk. However, no prospective study has evaluated whether weight loss actually affects adenoma recurrence. The aim of this study was to examine whether

Should bariatric surgery be offered for hepatocellular adenomas in obese patients?

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Background: Obesity and metabolic syndrome are increasingly recognized as risk factors for development of hepatocellular adenoma (HCA). The implications of weight loss on HCA regression has not been previously reviewed.

Lifestyle factors, obesity and the risk of colorectal adenomas in EPIC-Heidelberg.

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OBJECTIVE We investigated the association of lifestyle and obesity with colorectal adenoma risk in a prospective setting. METHODS At recruitment (1994-1998), information on diet, anthropometry, lifestyle, and medication was assessed in 25,540 participants of the EPIC-Heidelberg cohort. Until June

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