adrenocortical hyperfunction/ödem

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Cyclical edema and hypokalemia due to occult episodic hypercorticism.

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Yearly episodes of edema, hypokalemia, anxiety, and depression were found to be due to cortisol and deoxycorticosterone surges secondary to a pituitary adenoma in a woman without any of the usual clinical features of Cushing's syndrome. During the long clinical remissions, she had no recognizable

[Hypercorticism due to adrenal adenoma with glucorticoid and androgen hypersecretion, edema and hypokalemia, severe metabolic disorders].

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Endogenous hypercortisolism inducing reversible ocular hypertension.

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To describe the clinical findings of two patients with reversible ocular hypertension secondary to endogenous hypercortisolism.Retrospective, observational case series.A 65-year-old man (patient 1) and a 21-year-old woman

Pulmonary artery thrombosis in three dogs with hyperadrenocorticism.

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Varying degrees of respiratory distress developed in 3 dogs in which hyperadrenocorticism was diagnosed. The respiratory distress was attributed to pulmonary artery thrombosis. Radiography revealed pleural effusion, increased diameter and blunting of the pulmonary arteries, lack of perfusion of the

[Diabetes mellitus and massive lower leg edema without heart failure].

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BACKGROUND The 47-year-old male patient was admitted to the hospital because of newly diagnosed diabetes and elevated liver function tests (gamma glutamyl transferase 303 U/l). On admission the patient reported a reduction of appetite, which had increased during the past 2 weeks, fatigue, muscular

ISLET-CELL CARCINOMA (ZOLLINGER-ELLISON SYNDROME) WITH FULMINATING ADRENOCORTICAL HYPERFUNCTION AND HYPOKALEMIA.

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The production of ACTH-like material by tumours arising in non-endocrine tissue may initiate severe adrenocortical hyperfunction. The pathogenesis and clinical and laboratory features of Cushing's syndrome associated with such tumours are characteristic. The autonomous production by the tumour of

Significant hypercortisolism during fractionated radiotherapy in a patient with a large corticotroph adenoma: a case report and literature review.

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OBJECTIVE We describe a patient with a large, invasive corticotroph adenoma who developed severe hypercortisolism shortly after starting fractionated radiotherapy. METHODS We reviewed the patient's clinical course, along with relevant literature for similar reported cases. RESULTS A 29-year-old man

Hyperadrenocorticism of calorie restriction contributes to its anti-inflammatory action in mice.

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Calorie restriction (CR), which lengthens lifespan in many species, is associated with moderate hyperadrenocorticism and attenuated inflammation. Given the anti-inflammatory action of glucocorticoids, we tested the hypothesis that the hyperadrenocorticism of CR contributes to its attenuated

Hyperadrenocorticism, attenuated inflammation, and the life-prolonging action of food restriction in mice.

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Food restriction (FR), which extends life span, is associated with an enhanced diurnal elevation of glucocorticoids. This increase in glucocorticoids may contribute to longevity by chronically enhancing the same protective mechanisms mobilized during acute stress. The objective of this study was to

Cushing's disease cycling over ten years.

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Cycles of excessive cortisol secretion have been reported interposed with phases of remission lasting from few days to several months. In this study we report a ten year follow up on a patient who had four episodes of hypercortisolism and Cushing's syndrome associated with hypokalemia and edema and

Cyclic Cushing's disease with paradoxical response to dexamethasone.

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Cyclic Cushing's disease is an unusual disorder characterised by ACTH-dependent periodical increase of serum cortisol levels, clinically accompanied by peripheral edema, abnormalities of cardiac rhythm and hypokalemia. The condition may be unrecognised for years, since the typical features of

Enhanced Na+/H+ exchange in Cushing's syndrome reflects functional hypermineralocorticoidism.

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BACKGROUND Patients with Cushing's syndrome exhibit a bimodal distribution of maximal rates of the erythrocyte amiloride-sensitive Na+/H+ exchange (NHE). Enhanced erythrocyte NHE has recently been found in patients with primary aldosteronism. OBJECTIVE To test the hypothesis that occult

[Reversible metabolic syndrome].

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A 52-year-old male patient was admitted to the emergency department with dyspnea and hypertensive urgency. During the previous 6 months, the patient had noticed leg edema, weight gain (particularly in the face and abdomen), and impotence. 1.5 years ago, he was diagnosed with hypertension resistant

Cushing's syndrome patient who exhibited congestive heart failure.

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Cushing's Syndrome (CS) may sometimes lead to dilated cardiomyopathy, even though this condition can be partially or completely reversed after treatment. In this article we report the case of a 28-yr-old woman with CS secondary to adrenal adenoma who exhibited congestive heart failure as an initial

A patient with ectopic cortisol production derived from malignant testicular masses.

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BACKGROUND A 65-year-old man presented to an oncology clinic with bilateral testicular masses, lower extremity edema, and cushingoid appearance. METHODS Measurements of serum cortisol and adrenocorticotropic hormone levels, testicular ultrasound and abdominal CT scans, and review of histopathology

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