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Sida 1 från 322 resultat
The effect of cyanosis on perioperative platelet function as measured by multiple electrode aggregometry and postoperative blood loss in neonates and infants undergoing cardiac surgery.
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OBJECTIVE
Platelet dysfunction is one of the major haematological disturbances of cardiopulmonary bypass (CPB). In addition, cyanosis is known to cause further coagulation disturbances.
METHODS
We prospectively studied 110 children under 1 year of age for the effects of cyanosis on baseline platelet
Diagnosis and treatment of hereditary hemorrhagic telangiectasia in a pediatric patient with chronic cyanosis.
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Telangiectasis of the lungs, with two case reports of hereditary haemorrhagic telangiectasia with cyanosis.
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[Congenital cyanosis in a 6-year-old boy due to circumscribed lung angioma and hereditary hemorrhagic telangiectasis].
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Progressive cyanosis in a child with hereditary hemorrhagic telangiectasia (Osler-Weber-Rendu disease).
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Clinical pathologic conference no. 486 cardiac arrest, peripheral cyanosis, widespread bleeding, thrombocytopenia.
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Hepatogenous cyanosis and subarachnoid haemorrhage.
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Upper GI bleeding among neonates admitted to Mulago Hospital, Kampala, Uganda: a prospective cohort study.
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BACKGROUND
The World Health Organization (WHO) reports estimate that 85% of newborn deaths are due to infections, prematurity and fetal distress. These conditions are risk factors for upper GI bleeding (UGIB) in sick neonates. UGIB is associated with poor neonatal outcomes such as prolonged
[Hereditary hemorrhagic telangiectasia: a report of two cases]
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This article reports two children with hereditary hemorrhagic telangiectasia (HHT). Patient 1 was a boy aged 12 years and was admitted due to intermittent cough and wheezing for more than 10 years. This boy and his mother and grandmother had a history of epistaxis. The boy had a history of the
Perthes Syndrome associated with intramedullary spinal cord hemorrhage in a 4-year-old child: a case report.
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BACKGROUND
Perthes Syndrome (Traumatic asphyxia) is rare, which is caused by sudden compressive chest trauma and characterized by subconjunctival hemorrhage, facial edema, craniocervical cyanosis, and petechiae on the upper chest and face.
METHODS
We present the case of a 4-year-old Caucasian girl
[Exogenous endothelin-1 induced pulmonary hemorrhage in newborn rats and the antagonizing effect of calcitonin gene-related peptide].
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OBJECTIVE
To observe the phenomenon of pulmonary hemorrhage (PH) induced by exogenous endothelin-1 (exET-1) and the antagonizing effect of exogenous calcitonin gene-related peptide (exCGRP) in newborn rats.
METHODS
(1) To study the exET-1 induced PH: 100 newborn Wistar rats were randomly assigned
Hereditary hemorrhagic telangiectasia or Rendu-Osler-Weber syndrome in the same family.
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The authors present the case of three patients from the same family in whom hereditary hemorrhagic telangiectasia (HHT) or Rendu-Osler-Weber syndrome was diagnosed. The disease is rare and occurs with multiple telangiectases of the skin and mucosa, and pulmonary arteriovenous fistulae. The clinical
[Value of cyanosis and petechiae in strangulation and concurrent severe anemia (author's transl)].
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Cyanosis, petechiae, and hemorrhages into the soft tissues and muscles of the neck may be absent after strangulation (throttling) during life, if severe bleeding has happened before. If in severe anemia signs directly or indirectly caused by strangulation have developed, it may be suggested that
[Neonatal varicella: report of a case of bronchopneumonia and hemorrhagic pulmonary edema].
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We describe the case of a male newborn infant, whose mother developed varicella 20 days before delivery. At 2 hours of life the infant's general state suddenly deteriorated and he presented cyanosis, respiratory distress and emission of pink foam in his mouth. The infant was diagnosed with bilateral
Vitamin K deficiency--late onset intracranial haemorrhage.
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A retrospective study is presented of the clinical features and outcome of late onset haemorrhagic disease due to vitamin K deficiency in 11 babies who were admitted to the emergency or child neurology unit during a 4-year period (January 1994-December 1997). The disease occurred in infants between
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