myopia/trötthet

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Near vision stress: vergence adaptation and accommodative fatigue.

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Changes in the visual system following prolonged near work were investigated. Fifteen young, normal subjects undertook a severe, two hour long, binocular near visual task at 20 cm without any breaks. Fusional stress was assessed by near 'phoria change. Refractive change was measured with an

Instrument myopia conceptions, misconceptions, and influencing factors.

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The literature on instrument myopia is reviewed, with the review organized around three principal objectives. One is to describe and discuss various factors that may influence the degree to which instrument myopia is manifested, including monocular vs. binocular viewing, age, direction of focus,

Induced pupillary hippus following near vision: increased occurrence in visual display unit workers.

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Using a quasistatic method, it has been found previously that one-third of normal subjects retained pupillary constriction under empty-field conditions for a substantial period following a near-vision task. This pupil after-effect was dissociated from adaptation of tonic accommodation. In this

[Pathogenesis and treatment of accommodative disturbance].

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Using a computer-assisted infrared optometer with a pupillograph, we tried to obtain basic understanding of accommodative disturbance and its by investigating tonic (dark focus) level of accommodation and quasi-static accommodative response. In normal volunteers in whom either visual fatigue,

Case Studies in Neuroscience: Instability of the visual near triad in traumatic brain injury-evidence for a putative convergence integrator.

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Deficits of convergence and accommodation are common following traumatic brain injury, including mild traumatic brain injury, although the mechanism and localization of these deficits have been unclear and supranuclear control of the near-vision response has been incompletely understood. We describe

The development of topically acting carbonic anhydrase inhibitors as antiglaucoma agents.

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Inhibition of carbonic anhydrase (CA, EC 4.2.1.1) isoforms present in the eyes (CA I, II, IV and XII), with sulfonamides such as acetazolamide, methazolamide, ethoxzolamide and dichlorophenamide, is still widely used for the systemic treatment of glaucoma. The mechanism of action of these drugs

The development of topically acting carbonic anhydrase inhibitors as anti-glaucoma agents.

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Carbonic anhydrase inhibitors (CAIs) such as acetazolamide, methazolamide, ethoxzolamide and dichlorophenamide were and still are widely used systemic antiglaucoma drugs. Their mechanism of action consists in inhibition of CA isozymes present in ciliary processes of the eye (such as CA II, IV and

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