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necrobiotic disorders/hypoxia
Länken sparas på Urklipp
8 resultat
[Transcutaneous measurement of oxygen and carbon dioxide pressure in necrobiosis lipoidica].
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Transcutaneous measurement of oxygen pressure (PcO2) and carbon dioxide pressure (PcCO2) was performed in nine patients with histologically confirmed necrobiosis lipoidica. None of the patients had diabetes mellitus. All measurements were taken at the lower leg. In each case, the atrophic center,
Treatment of necrobiosis lipoidica diabeticorum by hyperbaric oxygen.
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Necrobiosis lipoidica diabeticorum is a chronic cutaneous complication of diabetes mellitus with microangiopathy as an important pathophysiologic factor. Because of the known success of hyperbaric oxygen in the treatment of chronic non-healing wounds, we used this mode of therapy to treat a diabetic
[Pathologic anatomy of traumatic shock during peace time].
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The study on the morphology of organs in 316 fatalities due to traumatic shock and blood loss and organs of 120 dogs and 25 rats sacrificed after reproduction of closed trauma of the chest accompanied by blood loss revealed the dependence of morphological changes on the time of shock development and
[Liver-damaging action of halothane--an animal experiment study].
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After long-term exhibition to halothane (up to 96 hours), centrolobular hepatic damage (fatty degeneration, necrobiosis and necrosis) developed in rats, proportionally to the number of anesthesias and shortening of the intervals. From these results no hepatotoxic effect of halothane can be derived,
Morphogenesis of cerebral matrix ectopies in human fetus and newborn.
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The subject of investigations was genesis of periventricular, cerebral matrix ectopies in human fetuses and newborns. The morphologic and histochemical analysis concerned the embryonal elements of the ventricular wall: namely neuroepithelium with membrana limitans externa and suporficial layer of
[Morphology and pathogenetic problems of the crush syndrome].
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Morphofunctional studies of muscles, heart, liver and kidneys after different periods of compression and decompression, as well as literature data indicate that crush syndrome is one of the most severe forms of traumatic shock. A wide range of pathologic effects of catecholamines and other
[Trigger mechanisms in the etiology of varicose and post-thrombophlebitic crural ulcers].
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The results of clinical study of 247 cases suffering from trophic crural ulcer are set forth in this paper. Their occurrence is mainly due to various diseases of deep, communicant and superficial veins (236 cases 95%). Venous hypertension in the deep veins, while the patient is standing, is the
Mechanisms and mediators in hepatic necrosis.
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Necrotic processes may be restricted to individual cell types of the liver or afflict several liver cells sequentially. Noxious agents may induce necrobiosis by different mechanisms of injury. In many instances, however, similar or identical terminal processes are involved, e.g. accumulation of Ca2+
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