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Sida 1 från 5063 resultat
Fever, tumor necrosis factor, and interleukin-6 in young, mature, and aged Fischer 344 rats.
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The purpose of this study was to compare the febrile responses of Fischer 344 rats of different ages [young (3-5 mo), mature (12-15 mo), and aged (24-27 mo; n = 8)] to two psychological stress paradigms, cage switch and exposure to an open field, as well as to injection of lipopolysaccharide (LPS).
Sterile necrosis of the liver due to primary epithelioid haemangio-endothelioma presenting as fever of undetermined origin.
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We report a case of primary epithelioid haemangio-endothelioma of the liver presenting as fever of undetermined origin. Due to the presence of iso-echoic sterile necrosis of the liver no abnormalities were found on four subsequent ultrasonographies of the liver. The definite diagnosis of
Avascular necrosis of the femoral head foreshadowing familial Mediterranean fever: apropos of three cases.
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We reported here on three patients in whom the diagnosis of familial Mediterranean fever was established after avascular necrosis of the femoral head had been detected. The pathogenesis and the management of this rare concomitance are discussed in the light of the relevant literature.
Fat necrosis of transverse colon and necrotizing pancreatitis in a patient with acute lymphoblastic leukemia (ALL): cause of massive ascites and high fever.
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We report a nine-year-old boy with acute lymphoblastic leukemia L3 (ALL-L3) and necrotizing pancreatitis in which necrosis was not limited to the pancreas. As our patient had a defective inflammatory process as a result of underlying malignant disease and neutropenia, the peripancreatic collection
An unusual complication of familial Mediterranean fever: intestinal volvulus and necrosis.
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Familial Mediterranean fever is an autosomal recessive disease characterized by recurring inflammatory attacks of synovial membranes. More than 95% of patients show peritoneal involvement which mimics acute abdomen and can sometimes cause unnecessary surgical intervention. The authors present two
FETAL HEPATIC NECROSIS IN GLANDULAR FEVER.
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A young man of 24 developed glandular fever, became jaundiced, and died in hepatic coma. At necropsy massive necrosis of the liver was found, thus emphasizing that all grades of severity of liver damage may occur in infectious mononucleosis.
Dermal necrosis associated with Rocky Mountain spotted fever in four dogs.
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Four dogs with Rocky Mountain spotted fever developed extensive dermal necrosis. Factors contributing to this complication included delay in initiation of appropriate therapy.
Two cases of fatal necrosis of the lesser pelvis in patients treated with combined radiotherapy and hyperthermia for cervical carcinoma.
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This study reports two cases of fatal necrosis of the lesser pelvis in patients with advanced cervical carcinoma, who had received combined radiotherapy and hyperthermia. The necrosis reached far from the high dose area, in one of the cases even outside the radiation portals. Both patients initially
Overlap syndrome between Familial Mediterranean fever and tumor necrosis factor receptor-associated periodic syndrome in a lupus patient.
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Autoinflammatory diseases represent an expanding spectrum of genetic and non-genetic inflammatory diseases characterized by recurrent episodes of fever and systemic inflammation, affecting joints, skin and serosal surfaces. Familial Mediterranean fever (FMF) is the most common autosomal recessive
Tumor necrosis factor in familial Mediterranean fever.
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OBJECTIVE
The pleiotropic inflammatory effects of tumor necrosis factor (TNF) prompted a study of this cytokine in familial Mediterranean fever (FMF), a recurrent polyserositis of unknown etiology.
METHODS
Thirty-six asymptomatic and 24 patients with acute FMF were studied and compared with 20
Effects of anti-tumor necrosis factor agents for familial mediterranean fever patients with chronic arthritis and/or sacroiliitis who were resistant to colchicine treatment.
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BACKGROUND
Effectiveness of anti-tumor necrosis factor (anti-TNF) agents in colchicine-resistant familial Mediterranean fever (FMF) patients has attracted attention in recent years.
OBJECTIVE
We analyzed the effect of anti-TNF agents on clinical findings of colchicine-resistant FMF patients with
Heat shock factor 1 protects mice from rapid death during Listeria monocytogenes infection by regulating expression of tumor necrosis factor alpha during fever.
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Heat shock factor 1 (HSF1) is a stress-induced transcription factor that promotes expression of genes that protect mammalian cells from the lethal effects of severely elevated temperatures (>42°C). However, we recently showed that HSF1 is activated at a lower temperature (39.5°C) in T cells,
Tumor necrosis factor-alpha and fever after peripheral inflammation in the rat.
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The involvement of endogenous tumor necrosis factor-alpha (TNF-alpha) in the pyrogenic [i.e., rise in colonic temperature (Tc)] and thermogenic [increase in oxygen consumption (VO2)] responses to inflammation was investigated in rats subjected to an intramuscular injection of turpentine. Turpentine
African swine fever virus infection induces tumor necrosis factor alpha production: implications in pathogenesis.
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We have analyzed the production of tumor necrosis factor alpha (TNF-alpha) induced by in vitro infection with African swine fever (ASF) virus (ASFV) and the systemic and local release of this inflammatory cytokine upon in vivo infection. An early increase in TNF-alpha mRNA expression was detected in
Increased production of nitric oxide in patients with hemorrhagic fever with renal syndrome--relation to arterial hypotension and tumor necrosis factor.
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In 15 consecutive subjects hospitalized with nephropathia epidemica, a European form of hemorrhagic fever with renal syndrome, the plasma concentrations of nitrate plus nitrite, stable metabolites of nitric oxide, were determined. From day 3 of onset of disease the concentrations increased, peak
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