norepinephrine/blödning

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Early norepinephrine infusion delays cardiac arrest after hemorrhagic shock in rats.

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BACKGROUND Severe hemorrhagic shock often results in cardiac arrest due to vital organ hypoperfusion, especially of the heart. Although fluid resuscitation is the mainstay of management in hemorrhagic shock, treatment of cardiac arrest in association with severe hemorrhagic shock is

Norepinephrine levels in experimental spinal cord trauma. Part 2: Histopathological study of hemorrhagic necrosis.

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Alpha methyl tyrosine (AMT) or reserpine administered intravenously 24 hours before sacrificed in the nontraumatized cat resulted in significant reduction in tissue levels of norepinephrine (NE) tested at the T-5 spinal cord level. Phenoxybenzamine given 2 hours before sacrifice did not alter NE

[Effect of elevated plasma norepinephrine on electrocardiographic changes in subarachnoid hemorrhage].

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We compared electrocardiographic abnormalities and plasma norepinephrine concentration in 40 patients with subarachnoid hemorrhage within the first 24 hours, at 72 hours, and after 1 week. In the 20 patients with high plasma norepinephrine concentrations within the first 24 hours, sinus tachycardia

Effect of elevated plasma norepinephrine on electrocardiographic changes in subarachnoid hemorrhage.

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We compared electrocardiographic abnormalities and plasma norepinephrine concentrations in 40 patients with subarachnoid hemorrhage within the first 24 hours, at 72 hours, and after 1 week. In 20 patients with high plasma norepinephrine concentrations within the first 24 hours, sinus tachycardia (p

Tissue levels of norepinephrine and epinephrine in hemorrhagic shock.

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Severe depletion of endogenous norepinephrine was observed in the brain, heart, liver, and spleen of albino rabbits in which hemorrhagic shock had been induced. On the other hand, the epinephrine content of these tissues was significantly elevated above the levels in tissues of control animals. The

Endogenous opiate peptides may limit norepinephrine release during hemorrhage.

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The involvement of the sympathetic nervous system in the cardiovascular response to hemorrhage and subsequent opiate receptor blockade was studied in conscious rabbits. Plasma catecholamines were measured by high-pressure liquid chromatography to indirectly assess sympathetic activity. Arterial

Effects of norepinephrine on dynamic variables of fluid responsiveness during hemorrhage and after resuscitation in a pediatric porcine model.

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BACKGROUND The effect of vasopressors on pulse pressure variation (PPV), stroke volume variation (SVV) and on the volumetric variable global end-diastolic volume (GEDV) during changing loading conditions is currently under debate. The aim of our study was to investigate the effect of norepinephrine

Effects of crystalloid and colloid resuscitation on hemorrhage-induced vascular hyporesponsiveness to norepinephrine in the rat.

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BACKGROUND We have shown previously that hemorrhagic hypotension is associated with a progressive development of vascular hyporeactivity to norepinephrine (NE). The present study investigated whether select crystalloid or colloid resuscitation fluids would ameliorate this

Intraperitoneal and intragastric norepinephrine instillation in massive gastrointestinal bleeding--clinical study. Preliminary report.

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Intraperitoneal and intragastric instillation of norepinephrine in high concentration can arrest bleeding of various origins in the gastrointestinal tract. Of the 21 cases studied, the hemorrhage was controlled in 80% of cases. This form of therapy is simple, prompt, safe and can be performed as a

Responses of ACTH, epinephrine, norepinephrine, and cardiovascular system to hemorrhage.

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Hemorrhages of various magnitudes were performed on conscious rats, and arterial pressure, heart rate, and plasma levels of adrenocorticotropin hormone (ACTH), epinephrine, and norepinephrine were measured. Eight rats were prepared with chronic femoral arterial cannulas and received a 10, 15, or 20

Thermal nociception potentiates the release of ACTH and norepinephrine by blood loss.

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Neuroendocrine and autonomic responses were assessed in chloralose-anesthetized cats after nociceptor activation and after acute blood loss, two of the sensory components that accompany injury. Plasma adrenocorticotropic hormone (ACTH), catecholamines (peripheral and adrenal), and cardiovascular

The effects of l-norepinephrine on myocardial zonal lesions in dogs in hemorrhagic shock.

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Intravenous l-norepinephrine was given to dogs in hemorrhagic shock to determine the effect on myocardial zonal lesions which are characteristically seen in hypovolemic shock. Zonal lesions produced by hemorrhagic shock in drug-treated and nondrug-treated groups were quantitatively compared both at

Differential concentration-dependent effects of prolonged norepinephrine infusion on intraparenchymal hemorrhage and cortical contusion in brain-injured rats.

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Under clinical conditions catecholamines are infused to elevate cerebral perfusion pressure and improve impaired posttraumatic cerebral microcirculation. This, however, is associated with the risk of additional hemorrhage in the acute phase following traumatic brain injury. In the present study we

Heart rate variability, norepinephrine and ECG changes in subarachnoid hemorrhage patients.

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While electrocardiographic abnormalities in subarachnoid hemorrhage patients have been known since 1947, the provocative mechanism has not yet been satisfactorily explained. The purpose of this study was to evaluate with some tests, the role of autonomic nervous system in provoking the

The role of norepinephrine and estradiol in the pathogenesis of cardiac wall motion abnormality associated with subarachnoid hemorrhage.

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OBJECTIVE The majority of patients with ventricular wall motion abnormality (WMA) associated with subarachnoid hemorrhage (SAH) are postmenopausal women. In addition to elevated catecholamine, the role of estrogen in the pathogenesis of WMA has recently been implicated. The objective of this study

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