orthostatic intolerance/stroke

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Contributions of MSNA and stroke volume to orthostatic intolerance following bed rest.

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We examined whether the altered orthostatic tolerance following 14 days of head-down tilt bed rest (HDBR) was related to inadequate sympathetic outflow or to excessive reductions in cardiac output during a 10- to 15-min head-up tilt (HUT) test. Heart rate, blood pressure (BP, Finapres), muscle

Does goal-directed fluid therapy affect postoperative orthostatic intolerance?: A randomized trial.

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BACKGROUND Early mobilization is important for postoperative recovery but is limited by orthostatic intolerance (OI) with a prevalence of 50% 6 h after major surgery. The pathophysiology of postoperative OI is assumed to include hypovolemia besides dysregulation of vasomotor tone. Stroke

Orthostatic intolerance after spaceflight.

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Orthostatic intolerance occurs commonly after spaceflight, and important aspects of the underlying mechanisms remain unclear. We studied 14 individuals supine and standing before and after three space shuttle missions of 9-14 days. After spaceflight, 9 of the 14 (64%) crew members could not complete

Predictors of orthostatic intolerance in healthy young women.

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OBJECTIVE Orthostatic intolerance is more prevalent in women. The purpose of this investigation was to evaluate the physiological responses of orthostatic tolerant and intolerant females to progressive lower body negative pressure (LBNP) and to identify predictors of orthostatic

Cardiovascular echographic and Doppler parameters for the assessment of orthostatic intolerance.

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OBJECTIVE To quantify the hemodynamic changes associated with orthostatic intolerance. METHODS The aortic flow, the cerebral and lower limb arterial flow and resistance, the flow redistribution between these two areas, and the femoral vein distension were assessed during two orthostatic tests

Cerebrovascular and cardiovascular responses associated with orthostatic intolerance and tachycardia.

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Idiopathic orthostatic intolerance syndrome is characterized by postural symptoms of cerebral hypoperfusion without arterial hypotension. Abnormal baroreceptor responses with deranged cerebral autoregulation leading to cerebral vasoconstriction have been proposed as a causative mechanism. The

Beta-blockade does not improve orthostatic intolerance induced by 20 days bed rest.

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To assess if propranolol influences orthostatic intolerance induced by prolonged bed rest (BR), a lower body negative pressure test (LBNP) and left ventricular (LV) echocardiography before and during -40mmHg of LBNP were performed with and without intravenous propranolol administration (0.04mg/kg)

Enhanced slow caudad fluid shifts in orthostatic intolerance after 24-h bed-rest.

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To evaluate mechanisms of late orthostatic intolerance, slow fluid shifts along the body axis were studied during deconditioning by 24-h bed-rest and during 13-min upright tilts before and after this manoeuvre. In 11 healthy male subjects the fluid volumes of a thorax and a calf segment (impedance

Orthostatic tachycardia with different onset time in patients with orthostatic intolerance.

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OBJECTIVE To investigate whether there is a difference in the neuro-autonomic and hemodynamic parameters in patients with postural tachycardia syndrome (POTS) according to onset time of orthostatic tachycardia during the Head-up tilt (HUT) test. METHODS We retrospectively reviewed neuro-autonomic

Simulated stand tests and centrifuge training to prevent orthostatic intolerance on Earth, moon, and Mars.

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One proposed method to overcome postflight orthostatic intolerance is for astronauts to undergo inflight centrifugation. Cardiovascular responses were compared between centrifuge and gravitational conditions using a seven-compartment cardiovascular model. Vascular resistance, heart rate, and stroke

Effects of rapid saline infusion on orthostatic intolerance and autonomic tone after 20 days bed rest.

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To test whether acute volume expansion can normalize orthostatic intolerance and autonomic tone after prolonged bed rest (BR), 23 men were subjected to 20 days BR. Left ventricular (LV) echocardiography was performed during the lower body negative pressure (LBNP) test before and after BR with and

Beat-to-beat analysis of pressure wave morphology for pre-symptomatic detection of orthostatic intolerance during head-up tilt.

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OBJECTIVE The aim of this study was to noninvasively define the hemodynamic profile characterizing the early response to tilting. BACKGROUND The mechanisms causing orthostatic intolerance have not been fully elucidated. Usually, patients undergoing tilt test are studied in a time-consuming way.

Autonomic activity and biomarker behavior in supine position and after passive postural stress in different orthostatic intolerance syndromes.

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OBJECTIVE Orthostatic intolerance (OI) syndromes are a confusing topic and determining a specific diagnosis to achieve optimal treatment can be troublesome. We sought to assess biomarker, hemodynamic and autonomic variables in OI patients (autonomic dysfunction [AD], postural orthostatic tachycardia

Orthostatic Intolerance After ISS and Space Shuttle Missions.

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Cardiovascular deconditioning apparently progresses with flight duration, resulting in a greater incidence of orthostatic intolerance following long-duration missions. Therefore, we anticipated that the proportion of astronauts who could not complete an orthostatic tilt test (OTT) would be higher on

Marked exacerbation of orthostatic intolerance after long- vs. short-duration spaceflight in veteran astronauts.

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OBJECTIVE The incidence of postflight orthostatic intolerance after short-duration spaceflight is about 20%. However, the incidence after long-duration spaceflight was unknown. The purpose of this study was to test the hypothesis that orthostatic intolerance is more severe after long-duration than

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