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Clinical Features of Ears With Otosclerosis and Endolymphatic Hydrops.

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Endolymphatic hydrops (EH) has been reported in ears with otosclerosis. The objective of this study was to investigate the clinical features of ears with otosclerosis and EH on magnetic resonance imaging (MRI) and identify predictors for the presence of

How Often Does Stapedectomy for Otosclerosis Result in Endolymphatic Hydrops?

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1) To evaluate the long-term (≥10 year) clinical incidence of endolymphatic hydrops (EH) after stapedectomy for otosclerosis, using low-frequency sensorineural hearing loss (LFSNHL) as a marker for EH. 2) To determine the histologic incidence of EH in human temporal bone specimens (TBS) with a

Otosclerosis and endolymphatic hydrops.

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It has been postulated that otosclerosis may produce vertigo by several mechanisms. One mechanism is by causing endolymphatic hydrops. We present six temporal bones in which otosclerosis and endolymphatic hydrops coexist. We consider that there is a spectrum-like interrelationship between these two

Cochlear and otoconial abnormalities in capsular otosclerosis with hydrops.

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Temporal bones from four patients with capsular otosclerosis were examined by microdissection. Otoconia and abnormal crystalline deposits were studied by scanning electron microscopy and x-ray analytical methods. One patient showed more or less symmetrical invasion of the basilar membrane and

Cochlear vascular pathology and hydrops in otosclerosis.

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Three ears with otosclerosis were found incidentally in a series of human temporal bones examined to evaluate cochlear sensorineural degeneration. Otosclerosis was identified with microdissection, surface preparation technique and transmission electron microscopy. Vascular abnormalities were present

Endolymphatic hydrops associated with otosclerosis.

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Endolymphatic hydrops (EH) associated with otosclerosis has been noted for many years. However, the causal relationship of these two entities remains controversial. Having reviewed the records of patients with otosclerosis describing fluctuant hearing loss and vertiginous symptoms, the authors found

Magnetic Resonance Imaging Evaluation of Endolymphatic Hydrops in Cases With Otosclerosis.

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OBJECTIVE Endolymphatic hydrops (EH) has been reported in cases with otosclerosis, and preoperative EH could be a risk factor for inner ear disturbances after stapes surgery. Visualization of EH has recently become possible using magnetic resonance imaging (MRI) with contrast agents. This study

Magnetic resonance imaging evaluation of endolymphatic hydrops andpost-operative findings in cases with otosclerosis.

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CONCLUSIONS The presence of endolymphatic hydrops (EH) in the vestibule on magnetic resonance imaging (MRI) might be a high-risk factor for complications in cases that are candidates for stapes surgery. OBJECTIVE Pre-operative detection of EH could be valuable in cases that are candidates for stapes

[Labyrinthine hydrops and otosclerosis; clinical base].

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Cochlear otosclerosis and endolymphatic hydrops.

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Postoperative complications of stapedectomy. Hydrops; perilymph fistula; regrowth of otosclerosis; prosthetic dislocations.

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Extensive otosclerosis and endolymphatic hydrops: histopathologic study of temporal bones.

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Asymptomatic Hydrops in a Case With Otosclerosis.

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A high jugular bulb and poor development of perivestibular aqueductal air cells are not the cause of endolymphatic hydrops in patients with Ménière's disease.

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OBJECTIVE The presence of endolymphatic hydrops in the inner ear, which can be detected with gadolinium-enhanced magnetic resonance imaging (Gd-MRI), is widely recognized as the main pathological cause of Ménière's disease (MD). However, the precise mechanisms underlying the development of

Otosclerosis associated with Ménière's disease: a histological study.

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A histological study of a pair of temporal bones was performed in a case of Menière's disease. A severe endolymphatic hydrops and extensive capsular otosclerosis bilaterally was found. Severe endolympathic hydrops results from otosclerotic endolympahtic duct occlusion. Our unique histopathological

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