papilledema/hypoxia

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Protective effects of dexamethasone on hypoxia-induced retinal edema in a mouse model.

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Hypoxia-induced retinal edema primarily induced by vascular lesion is seen in various conditions such as diabetic retinopathy (DR) and retinal vein occlusion (RVO). The edematous changes in these conditions occur mainly in intermediate and deep layers of retina as a result of disruption of the inner

Effects of avastin on expression of AQP4 in Müller cells under hypoxia.

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The aim of this study was to investigate the effects of Avastin on aquaporin4 (AQP4) expression in human retinal Müller cells in vitro under hypoxia, so as to explore the mechanism of Avastin treating retinal edema. The human Müller cells were cultured using the enzymatic digestion method. Müller

Experimental retinal branch vein occlusion in miniature pigs induces local tissue hypoxia and vasoproliferative microangiopathy.

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In miniature pigs, retinal veins were experimentally occluded using argon laser coagulation. Microvascular modifications leading to retinal hemorrhages and retinal edema were observed some hours after the occlusion. These lesions resolved progressively within 3 weeks after the occlusion, but in most

microRNA-320a prevent Müller cells from hypoxia injury by targeting aquaporin-4

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Müller cells are closely related to diabetic retinopathy (DR). Aquaporin-4 (AQP4) can effectively promote the diffusion of water across cellular membranes. However, the dynamic balance of water plays key role in many diseases, such as cerebral edema. Meanwhile, the unusual expression and

In Vivo Imaging of Retinal Hypoxia Using HYPOX-4-Dependent Fluorescence in a Mouse Model of Laser-Induced Retinal Vein Occlusion (RVO).

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To demonstrate the utility of a novel in vivo molecular imaging probe, HYPOX-4, to detect and image retinal hypoxia in real time, in a mouse model of retinal vein occlusion (RVO). Retinal vein occlusion was achieved in adult mice by photodynamic retinal vein thrombosis (PRVT). One or two major

Apelin supports primary rat retinal Müller cells under chemical hypoxia and glucose deprivation.

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Müller cells support the integrity of the blood-retinal barrier, whereas their dysfunction under pathological conditions may contribute to retinal edema formation. The apelin peptide, as the endogenous ligand of G protein-coupled receptor APJ, participates in numbers of physiological and

Hypoxia-ischemia and retinal ganglion cell damage.

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Retinal hypoxia is the potentially blinding mechanism underlying a number of sight-threatening disorders including central retinal artery occlusion, ischemic central retinal vein thrombosis, complications of diabetic eye disease and some types of glaucoma. Hypoxia is implicated in loss of retinal

Papilledema and obstructive sleep apnea syndrome.

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OBJECTIVE To characterize the pathogenesis and clinical features of optic disc edema associated with obstructive sleep apnea syndrome (SAS). METHODS A series of 4 patients with SAS and papilledema (PE) underwent complete neuro-ophthalmologic evaluation and lumbar puncture. In 1 patient, continuous

High altitude retinal hemorrhage: a clinical and pathological case report.

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Retinal hemorrhages may occur at high altitudes. They occur more commonly in association with acute mountain sickness and particularly with high-altitude cerebral edema. We describe a 27-year-old man who experienced retinal hemorrhages as well as papilledema and coma at 5,330-m altitude and who died

Origins of Cerebral Edema: Implications for Spaceflight-Associated Neuro-Ocular Syndrome.

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Spaceflight-associated neuro-ocular syndrome (SANS) was first described in 2011 and is associated with structural ocular changes found to occur in astronauts after long-duration missions. Despite multiple insufficient potential terrestrial models, an understanding of the etiology has

[High altitude retinopathy after Himalayan ascent].

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BACKGROUND Visual disturbances after high altitude exposure were first reported in 1969. Later, the term "High Altitude Retinal Hemorrhage-HARH" has been used for the ensuing retinal hemorrhages and vascular engorgement. METHODS A 31-year-old Caucasian male presented to our outpatient department 1

[The fundus oculi in chronic anemias].

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The investigation was carried out in 168 cases thus selected as chronic anaemia to be the only affection. In 49 patients (20%) the following changes, single or associated, were found: retinal pallor in 15% of the cases, superficial retinal bleedings in 6%, deep bleedings in 3.5%, retinal exudates in

Disk edema in an overweight woman.

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A 46-year-old extremely obese black woman presented with headaches, blurred vision, and visual obscurations. Her exam was notable for bilateral severe papilledema, retinal hemorrhages, and lethargy. Her CAT scan was normal, and a spinal tap revealed a very high opening pressure. Although this

Sleep-disordered breathing and effects on ocular health.

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Sleep-disordered breathing may make the eye vulnerable due to the direct effect of hypoxia or it can involve pathways that lead to impaired autoregulation of optic nerve perfusion. In this review, we discuss our present understanding of the interactions that occur between sleep-disordered breathing

Restoring retinal neurovascular health via substance P.

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Regulation of vascular permeability plays a major role in the pathophysiology of visually threatening conditions such as retinal vein occlusion and diabetic retinopathy. Principally, several factors such as vascular endothelial growth factor (VEGF), are up-regulated or induced in response to hypoxia

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