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phospholipid/seizures
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Sida 1 från 153 resultat
Brain levels of N-acylethanolamine phospholipids in mice during pentylenetetrazol-induced seizure.
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The N-acylethanolamine phospholipids (NAPE) are precursors for N-acylethanolamines (NAE), including anandamide (20:4-NAE), which is a ligand for the cannabinoid receptors. Previously, NAPE were believed to be found only in injured tissue, e.g., after neurodegenerative insults. Neuronal injury may
[Metabolism of neuronal and neuroglial phospholipids in seizures].
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Metabolism of individual phospholipids was studied in neurones and neuroglia under conditions of convulsions caused by picrotoxin administration. Metabolic activity of all the phospholipids studied was increased in neuronal membranes under extreme conditions of convulsive attacks. The rate of
[Blood and cerebrospinal fluid phospholipid composition in children with a syndrome of febrile and epileptic convulsions].
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Children with febril and epileptic convulsion during different periods of the disease demonstrate changes in the correlation of phospholipids in the blood plasma. These changes are mainly of a general character and do not depend upon the genesis of convulsions. The largest differences were seen in
The Drosophila easily shocked gene: a mutation in a phospholipid synthetic pathway causes seizure, neuronal failure, and paralysis.
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We have characterized easily shocked (eas), a Drosophila "band-sensitive" paralytic mutant. Electrophysiological recordings from flight muscles in the giant fiber pathway of adult eas flies reveal that induction of paralysis with electrical stimulation results in a brief seizure, followed by a
GABA and phospholipids in penicillin-induced seizures.
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The effect of a suspension of GABA and phosphatidylserine (PS), phosphatidylcholine, or phosphatidylethanolamine was studied on penicillin-induced epileptic activity in rats. GABA-PS significantly reduced the number of spikes, in comparison with either the other phospholipid compounds or normal
The effect of pyridoxal phosphate-induced convulsive seizures on rat brain phospholipid metabolism.
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The intraventricular injection of pyridoxal phosphate (PLP; 1 mumole/brain) to rats causes convulsive seizures beginning 3 min after injection and lasting for about 20 min. The incorporation of [2-3H] glycerol into rat brain glycerides has been studied to ascertain whether treatment with PLP affects
Characteristic features of phospholipid metabolic disorders in membranes of erythrocytes of white rats in corazolum-induced epileptoid seizures.
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Biochemical studies on convulsions, with special reference to the relation between convulsive activity and metabolic changes in brain phospholipids and ATP.
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Studies on the brain phospholipids. II. The effect of electrically induced convulsions on the incorporation of P32 into rabbit brain phospholipids.
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The metabolism of individual phospholipids in the rat brain during hypoglycaemia, anaesthesia and convulsions.
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Effects of corticotropin and various convulsion-inducing agents on the P32 content of brain phospholipids, nucleoproteins and total acid-soluble phosphorus compounds.
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High levels of glycine and serine as a cause of the seizure symptoms of cavernous angiomas?
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Cavernous angiomas are vascular malformations that cause neurodegeneration and symptoms including epileptiform seizures, headache, and motor deficits. Following neurosurgical removal of the angiomas, patients mostly recover well and become seizure-free. This study reports on the levels of certain
[A case of anti-phospholipid antibody syndrome associated with subdural hematoma].
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We report a case of a 47-year-old man with anti-phospholipid antibody syndrome associated with subdural hematoma. The patient had several episodes of arthritis during his thirties. He developed venous thrombosis in his right lower leg at the age of 35, when laboratory studies demonstrated
The relationship between lupus anticoagulants and antibodies to phospholipid.
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The relationship between lupus anticoagulants and antibodies directed against negatively charged phospholipids, as measured by an enzyme-linked immunosorbent assay, was explored in a series of plasma samples from 100 patients with well-characterized lupus anticoagulants. Only 73% of the patients had
Synergistic effect of phosphatidylserine with gamma-aminobutyric acid in antagonizing the isoniazid-induced convulsions in mice.
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The influence of phosphatidylserine (PS) on the isoniazid-induced convulsions has been studied in mice. Sonicated dispersions of this phospholipid given intravenously do not show anticonvulsant activity but they do so when gamma-aminobutyric acid (GABA) is simultaneously injected. GABA alone is
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