placental insufficiency/fetma

Länken sparas på Urklipp

Välj sorteringstyp

Sida 1 från 51 resultat

Uteroplacental insufficiency increases visceral adiposity and visceral adipose PPARgamma2 expression in male rat offspring prior to the onset of obesity.

Endast registrerade användare kan översätta artiklar

Logga in Bli medlem

Uteroplacental insufficiency (UPI) induced intrauterine growth restriction (IUGR) predisposes individuals to adult onset metabolic morbidities, including insulin resistance and cardiovascular disease. An underlying component of the development of these morbidities is adipose dysfunction;

Maternal Obesity and Male Genital Anomalies: Potential Role of Placental Insufficiency and Metabolic Syndrome.

Endast registrerade användare kan översätta artiklar

Logga in Bli medlem

β2-Adrenergic receptor desensitization in perirenal adipose tissue in fetuses and lambs with placental insufficiency-induced intrauterine growth restriction.

Endast registrerade användare kan översätta artiklar

Logga in Bli medlem

Placental insufficiency-induced intrauterine growth restriction (IUGR) fetuses have chronic hypoxaemia and elevated plasma catecholamine concentrations. In this study, we determined whether adrenergic responsiveness becomes desensitized in the perirenal adipose tissue of IUGR fetuses and lambs by

Preeclampsia, placental insufficiency, and autism spectrum disorder or developmental delay.

Endast registrerade användare kan översätta artiklar

Logga in Bli medlem

OBJECTIVE Increasing evidence suggests that autism spectrum disorder (ASD) and many forms of developmental delay (DD) originate during fetal development. Preeclampsia may trigger aberrant neurodevelopment through placental, maternal, and fetal physiologic mechanisms. OBJECTIVE To determine whether

Catecholamines mediate multiple fetal adaptations during placental insufficiency that contribute to intrauterine growth restriction: lessons from hyperthermic sheep.

Endast registrerade användare kan översätta artiklar

Logga in Bli medlem

Placental insufficiency (PI) prevents adequate delivery of nutrients to the developing fetus and creates a chronic state of hypoxemia and hypoglycemia. In response, the malnourished fetus develops a series of stress hormone-mediated metabolic adaptations to preserve glucose for vital tissues at the

Placental insufficiency contributes to fatty acid metabolism alterations in aged female mouse offspring.

Endast registrerade användare kan översätta artiklar

Logga in Bli medlem

Intrauterine growth restriction (IUGR) is an accepted risk factor for metabolic disorders in later life, including obesity and type 2 diabetes. The level of metabolic dysregulation can vary between subjects and is dependent on the severity and the type of IUGR insult. Classical IUGR animal models

Fetal undernutrition, placental insufficiency and pancreatic β-cell development programming in utero.

Endast registrerade användare kan översätta artiklar

Logga in Bli medlem

The prevalence of obesity and type 2 (T2D) diabetes is a major health concern in the United States and around the world. T2D is a complex disease characterized by pancreatic β-cell failure in association with obesity and insulin resistance in peripheral tissues. Although several genes associated

Impact of maternal obesity on fetal programming of cardiovascular disease.

Endast registrerade användare kan översätta artiklar

Logga in Bli medlem

The in utero environment is a key determinant of long-term health outcomes; poor maternal metabolic state and placental insufficiency are strongly associated with these long-term health risks. Human epidemiological studies link maternal obesity and offspring cardiovascular disease in later life, but

Tacrolimus in the prevention of adverse pregnancy outcomes and diabetes-associated embryopathies in obese and diabetic mice.

Endast registrerade användare kan översätta artiklar

Logga in Bli medlem

T2DM is a high-risk pregnancy with adverse fetal and maternal outcomes including repeated miscarriages and fetal malformations. Despite the established association between placental insufficiency and poor maternal Th1-adaptability to the development of pregnancy complications in T2DM, there have

Maternal diet-induced obesity and offspring cardiovascular health.

Endast registrerade användare kan översätta artiklar

Logga in Bli medlem

It is widely recognized that environmental insults during adulthood including smoking, lack of exercise and a poor diet increases an individual's risk of cardiovascular disease (CVD). However, research initiated over the last two decades has highlighted that our risk of CVD can be programmed

Exposure to uteroplacental insufficiency reduces the expression of signal transducer and activator of transcription 3 and proopiomelanocortin in the hypothalamus of newborn rats.

Endast registrerade användare kan översätta artiklar

Logga in Bli medlem

IUGR has been linked to the development of type 2 diabetes. Recent data suggest that some of the molecular defects underlying type 2 diabetes reside in the CNS. Disruption of the signal transducer and activator of transcription 3 (STAT3) in the hypothalamic neurons expressing leptin receptor,

The association of maternal obesity with fetal pH and base deficit at cesarean delivery.

Endast registrerade användare kan översätta artiklar

Logga in Bli medlem

OBJECTIVE To evaluate the association between maternal body mass index (BMI) and umbilical cord acid-base status at the time of cesarean delivery. METHODS We conducted a retrospective multicenter cohort study using data from the Cesarean Section Registry of the Eunice Kennedy Shriver National

Neonatal management and long-term sequelae.

Endast registrerade användare kan översätta artiklar

Logga in Bli medlem

Intrauterine or fetal growth restriction is best defined by using customised birth weight percentiles based upon the growth potential for an individual infant. Growth restriction in utero may be classified as asymmetric or symmetric depending upon the duration of the process. Asymmetric growth

A triple risk model for unexplained late stillbirth.

Endast registrerade användare kan översätta artiklar

Logga in Bli medlem

BACKGROUND The triple risk model for sudden infant death syndrome (SIDS) has been useful in understanding its pathogenesis. Risk factors for late stillbirth are well established, especially relating to maternal and fetal wellbeing. CONCLUSIONS We propose a similar triple risk model for unexplained

Genetic selection of embryos that later develop the metabolic syndrome.

Endast registrerade användare kan översätta artiklar

Logga in Bli medlem

THE BARKER HYPOTHESIS: Is an excellent explanation of the process where human and animal foetuses exposed to malnutrition, either by maternal malnutrition or placental insufficiency, are metabolically programmed, with selective stunting of cell differentiation and organ growth. With the postnatal

Tidigare
1
2
3
4
Nästa

Gå med på vår
facebook-sida

Herbal & Natural Medicine

Den mest kompletta databasen med medicinska örter som stöds av vetenskapen

Fungerar på 55 språk
Växtbaserade botemedel som stöds av vetenskap
Örter igenkänning av bild
Interaktiv GPS-karta - märka örter på plats (kommer snart)
Läs vetenskapliga publikationer relaterade till din sökning
Sök efter medicinska örter efter deras effekter
Organisera dina intressen och håll dig uppdaterad med nyheterna, kliniska prövningar och patent

Skriv ett symptom eller en sjukdom och läs om örter som kan hjälpa, skriv en ört och se sjukdomar och symtom den används mot.
* All information baseras på publicerad vetenskaplig forskning