polyarteritis nodosa/phosphatase

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Trichinosis-related polyarteritis nodosa.

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During an outbreak of trichinosis, two young men--one with established trichinosis and the other with suspected infection--were found to have clinical, radiologic and histologic stigmata of a systemic necrotizing vasculopathy equivalent to classic polyarteritis nodosa. The parasitosis manifested as

Polyarteritis nodosa of the liver: a report of two cases.

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Two patients with liver disease due to polyarteritis nodosa are described. They presented in a similar manner, with a swinging fever, a polymorphonuclear leucocytosis and high alkaline phosphatase levels, but the natural history of the illness was different, with revocery in one and death in the

Immunohistochemical characterization of inflammatory cells and immunologic activation markers in muscle and nerve biopsy specimens from patients with systemic polyarteritis nodosa.

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OBJECTIVE To investigate the phenotype of infiltrating cells in classic lesions of polyarteritis nodosa (PAN). METHODS Twenty-one muscle and 10 sural nerve biopsy samples from 24 patients with systemic PAN were studied using avidin-biotin-peroxidase and alkaline phosphatase-anti-alkaline phosphatase

[Menetrier's hypertrophic gastritis associated with polyarteritis nodosa].

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Case presentation of a 61 year old male who presented with a 3 months' history of anorexia, weight loss, prolonged febrile syndrome, myalgias, peripheral polyneuropathy, abdominal pain, anemia, renal insufficiency, sonographically detected splenomegaly, elevated alkaline phosphatase,

Neuromuscular manifestations of L-tryptophan-associated eosinophilia-myalgia syndrome: a histomorphologic analysis of 14 patients.

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The recent delineation of a clinical syndrome marked by eosinophilia, myalgia, and scleroderma-like skin changes associated with L-tryptophan use has necessitated the Centers for Disease Control to initiate a health alert. The likely association of L-tryptophan ingestion with a syndrome that mimics

[Systemic vasculitis as a cause of fever of unknown origin].

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A prospective clinical study (1974-1988) was carried out in 33 patients with several types of systemic vasculitis (SV) presenting as fever of unknown origin (FUO) according to the 1961 Petersdorf and Beeson's criteria. Histological confirmation, either from biopsy or necropsy, was available in all

Clinical features of liver disturbance in rheumatoid diseases: clinicopathological study with special reference to the cause of liver disturbance.

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BACKGROUND Liver disturbance in rheumatoid diseases results not only from liver disease associated with the rheumatoid diseases themselves but also from various other causes. This study aimed to elucidate the clinical features of liver disturbance in rheumatoid diseases, focusing on the cause of

The dermatopathologic manifestations of hepatitis C infection: a clinical, histological, and molecular assessment of 35 cases.

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Cutaneous eruptions related to hepatitis C virus (HCV), a major cause of hepatitis in the setting of blood transfusion, intravenous drug abuse, organ transplantation, and hemodialysis, are typically reported as isolated cases. We encountered 35 cases of HCV infection associated with cutaneous

Extrahepatic autoimmune diseases in primary biliary cholangitis: prevalence, significance for clinical presentation and disease outcome

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Background: The prevalence and clinical significance of extrahepatic autoimmune diseases (EHAID) have not been evaluated in a large cohort of primary biliary cholangitis (PBC). Methods:

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