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Sida 1 från 30 resultat
Changes in red cell, plasma, inulin, and total water compartments in rat femurs. Comparison of vitamin D deficiency and diphosphonate induced rickets.
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The vascular space, as measured with the use of 99m-technetium-labelled red cells and 131I-serum albumin, as well as the 14C-inulin and 3H2O fluid compartments were measured in rats which had been made rachitic by the use of a low phosphate vitamin D-deficient diet. A similar group of rachitic
[Aspects of juvenile rickets/osteopenia in black children].
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Juvenile rickets or osteopenia in rural black children is thought to be due to low calcium intake. Characteristic findings include mild calcium deficiency, normal serum phosphate levels, increased alkaline phosphatase concentrations and normal plasma vitamin D levels. The present series consisted of
Effects of hydrochlorothiazide and amiloride in renal hypophosphatemic rickets.
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The effects of thiazide diuretics on serum phosphate concentration, renal tubular threshold for phosphate, and urinary calcium excretion in children with renal hypophosphatemic rickets were studied. There were nine controlled acute studies conducted in five patients, and, in addition, seven
[Vitamin D3 overdosage due to rashly diagnosed rachitis in a child with distal tubular acidosis].
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Metabolic acidoses are diseases causing many diagnostic and therapeutic problems. Compensated metabolic acidosis can be unrecognised for a long time. This refers especially to isolated renal tubular acidosis (RTA). Unrecognised RTA causes calcium and phosphorus balance disturbances with clinical
Effect of consumption of food cooked in aluminium or stainless-steel pots on Bangladeshi children with calcium-deficient rickets: an eight month trial.
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The putative role of aluminium intake in young Bangladeshi children (1.5 to 4 years of age) with calcium-deficient rickets was evaluated in a non randomised controlled eight month trial. The effects of aluminium or stainless-steel cooking pots on bone metabolism were assessed by measuring blood
Case report of an infant with severe vitamin D deficiency rickets manifested as hypocalcemic seizures.
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BACKGROUND
Hypocalcemic seizures are uncommon in the post-neonatal period. We report an infant with hypocalcemic seizures caused by severe deficiency of vitamin D.
METHODS
A five-month-old male infant was admitted to hospital in March 2013 with recurrent generalized afebrile seizures resistant to
Rickets in premature infants fed different formulas.
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To study the role of formula as a cause of rickets, we randomly assigned 46 very-low-birth-weight (VLBW) infants (less than or equal to 1,500 gm) to one of three groups receiving either Isomil, a soy isolate formula, Similac with Iron, a common milk-based formula, or Similac 24 LBW, a hypercaloric
Comparisons of oral calcium, high dose vitamin D and a combination of these in the treatment of nutritional rickets in children.
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Nutritional rickets remains a common child health problem in Turkey and many other developing countries. Although vitamin D deficiency is accepted as the basic problem underlying the disease, others postulate that a deficiency of dietary calcium, rather than vitamin D, is often responsible for the
Hypophosphatemic rickets: A case of recurrent pathological fractures.
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BACKGROUND
Renal phosphate-wasting disorders are the most common form of hereditary rickets and osteomalacia in western countries, but are rarely reported in India. Therefore, we report here a case of hypophosphatemic rickets.
OBJECTIVE
To report a case of hypophosphatemic rickets presenting with
[Lipids bound with blood plasma proteins in normal state and in experimental rachitis].
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The lipid composition of transport and specific proteins was studied in normal state and with experimental rachitis. The amount of phospholipids in blood plasma proteins control animals is established to decrease as follows: albumins, immunoglobulins, alpha, beta-globulins, fibrinogen. The bulk of
EXPERIMENTAL RICKETS IN RATS : V. THE EFFECT OF VARYING THE ORGANIC CONSTITUENTS OF A RICKETS-PRODUCING DIET.
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1. Casein phosphorus does not completely prevent the development of rickets when substituted in Diet 84 in amount equivalent to a protective dose of basic potassium phosphate. 2. The protection given by lecithin is equivalent to its phosphorus content. 3. The protection given by yeast is at least
Rickets and dysmorphic findings in a child with abetalipoproteinemia.
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Abetalipoproteinemia (ABL) is characterized by acanthocytosis, hypocholesterolemia, and steatorrhea. Here, we describe a case of ABL associated with rickets and dysmorphic findings and the subsequent therapeutic course in an 18-month-old male referred for evaluation for failure to thrive and chronic
Prevalence of vitamin D deficiency and rickets in children with cholestasis in Iran.
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This study was aimed to determine prevalence of vitamin D deficiency and rickets in children with cholestatic liver diseases. Forty eight children with established cholestatic liver disease who referred to gastrointestinal clinic of Children Medical Center (Tehran, Iran) between April 2010 and March
Noncollagenous bone proteins in experimental rickets in the rat.
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Rats were raised in the absence of vitamin D in utero and throughout post-fetal life and neither 1,25-dihydroxyvitamin D3 nor related metabolites were detected in serums. No changes were observed in the relative amount of extractable noncollagenous bone proteins (NCP) in rachitic compared to
Vitamin D nutrition in pregnant women at term and in newly born babies in Saudi Arabia.
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A survey to assess the vitamin D nutritional state in 119 pregnant women at term and in their newborns was undertaken in Riyadh, Saudi Arabia. Concentrations of 25-hydroxy vitamin D (25-(OH)D) were below 4 ng/ml in 30 of 119 maternal sera, in 11 of which they were undetectable. The median
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