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sialadenitis/carbohydrate
Länken sparas på Urklipp
5 resultat
Serum and histological IgG4-negative type 1 autoimmune pancreatitis.
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A 66-year-old man who was on oral medication for type 2 diabetes experienced a rapid decline in glycemic control (increase in glycosylated hemoglobin level from 7.7 to 10.2% over 3 months). Abdominal ultrasonography revealed a 20-mm hypoechoic mass in the pancreatic tail. Serum tumor marker
Salivary and oral components of Sjögren's syndrome.
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The intraoral symptoms and signs of SS are not specific to SS, being shared with other conditions in which salivary function is diminished. The decrease in saliva causes chronic oral discomfort and functional problems and predisposes patients to dental caries and oral candidiasis. Many methods have
[The mechanisms of disordered oxidative metabolism at different stages of acute experimental inflammation of the salivary glands].
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Acute purulent sialadenitis was modelled in experiments on rabbits by injecting Staphylococcus aureus. Follow-up of the development of inflammation showed an intimate relation between changes of the level of gland oxygenation and the values characterizing tissue respiration, carbohydrate metabolism,
Exacerbation of experimental autoimmune encephalomyelitis in mice deficient for DCIR, an inhibitory C-type lectin receptor.
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Dendritic cell immunoreceptor (DCIR) is a C-type lectin receptor containing a carbohydrate recognition domain in its extracellular portion and an immunoreceptor tyrosine–based inhibitory motif, which transduces negative signals into cells, in its cytoplasmic portion. Previously, we showed that
Dcir deficiency causes development of autoimmune diseases in mice due to excess expansion of dendritic cells.
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The dendritic cell immunoreceptor (official gene symbol Clec4a2, called Dcir here) is a C-type lectin receptor expressed mainly in dendritic cells (DCs) that has a carbohydrate recognition domain in its extracellular portion and an immunoreceptor tyrosine-based inhibitory motif, which transduces
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