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stomach ulcer/nikotin
Länken sparas på Urklipp
Sida 1 från 204 resultat
Peptic ulcer disease and the tar and nicotine yield of currently smoked cigarettes.
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In order to examine the association of smoking cigarettes with a lower tar and nicotine yield to peptic ulcer disease, a common disease associated in past studies with cigarette smoking. We used questionnaire information provided by 9009 current regular cigarette smokers. The percentage who ever had
Natural history of peptic ulcer disease in Poland. Part IV. Dietary habits, mode of eating, coffee, tea, tobacco and alcohol, addiction's.
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In the "Ulcer" research programme 5813 male and female patients with peptic ulcer of the upper gastrointestinal tract were analysed paying attention to the quality of their diet and mode of meal taking. Weight loss was found to occur in these patients and information was gathered concerning alcohol,
Tobacco use as contributory factor in peptic ulcer disease.
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OBJECTIVE
To find out the association of tobacco as a contributory factor in patients presenting with peptic ulcer disease (PUD).
METHODS
A descriptive case study.
METHODS
Department of Medicine, Postgraduate Medical Institute, Lady Reading Hospital, Peshawar, from May 2001 to April 2002.
METHODS
A
Effects of nicotine on activity and stress-induced gastric ulcers in rats.
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Nicotine is known to influence locomotor activity. The alkaloid also intensifies gastric ulcer formation in stressed rats. The effects of nicotine on locomotor activity in relation to gastric lesions induced by restraint at 4 degrees C for 2 h (stress) were, therefore, studied. Ten-day treatment
Tobacco cigarette smoke aggravates gastric ulcer in rats by attenuation of ulcer margin hyperemia.
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We tested the hypothesis that attenuation of the hyperemia at the margin of acetic acid-induced gastric ulcer in rats by tobacco cigarette smoke will increase the size of the ulcer in the acute and the healing stages. Compared with the adjacent mucosa, blood flow measured by hydrogen gas clearance
Nicotine and gastric ulcers in stress.
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Chronic nicotine treatment worsens stomach mucosal damage by cold (4 degrees C) and restraint (stress): it dose- and time-dependently intensifies stress-evoked gastric glandular ulceration, mast cell degranulation and motility. Nicotine 50 micrograms/ml drinking water, given ad libitum to female
Peptic ulcer and mental disorders among adults in the community: the role of nicotine and alcohol use disorders.
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OBJECTIVE
Previous studies have documented links between peptic ulcer disease (PUD) and mood and anxiety disorders among adults in the community. Several substance use disorders (e.g., nicotine and alcohol dependence) are highly comorbid with mood/anxiety disorders and have been also linked with
The effect of nicotine on ethanol-induced gastric ulcers in rats.
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Nicotine, in concentrations of 5 and 25 micrograms/ml drinking water, given ad libitum for 10 days, dose-dependently increased lesion formation and worsened ethanol-induced ulceration in rat stomachs. Daily fluid intake and b.wt gain were not adversely affected by nicotine pretreatment.
[Effects of nicotine, alcohol and caffeine on the incidence, healing and recurrence rate of peptic ulcer].
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A causal relationship exists between the incidence of peptic ulcer and smoking: smoking is significantly associated with a higher prevalence of gastric duodenal ulcer. Either type of peptic ulcer demonstrates a lower healing rate in smokers as compared to non-smokers and impaired spontaneous and
[Tobacco and alcohol are risk factors of complicated peptic ulcers. A prospective cohort study].
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Both the incidence of and mortality from bleeding and perforated peptic ulcers are growing. We assessed the association between smoking, ingestion of alcohol (including the type of alcoholic beverage), and risk of a complicated peptic ulcer in a population-based study of 26,518 Danish subjects
[Meta-analysis of the prevalence of peptic ulcer in relation to tobacco consumption].
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The reported studies of the association between smoking and the incidence of peptic ulcer have had nonconclusive results. Therefore, we have carried out a quantitative review including and evaluating all the factors related to that association. To this end, the techniques of meta-analysis suggested
[Nicotine & peptic ulcer].
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Tobacco and Peptic Ulcers.
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[Nicotine and peptic ulcer].
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Smoking and gastric ulcers: the possible role of nicotine.
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