alpha fructose/hypoxia

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Mitochondrial localization of TIGAR under hypoxia stimulates HK2 and lowers ROS and cell death.

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Ingia / Ingia

The p53-inducible protein TIGAR (Tp53-induced Glycolysis and Apoptosis Regulator) functions as a fructose-2,6-bisphosphatase (Fru-2,6-BPase), and through promotion of the pentose phosphate pathway, increases NADPH production to help limit reactive oxygen species (ROS). Here, we show that under

[Isolation of a novel fructose-1,6-bisphosphate aldolase gene from Codonopsis lanceolata and analysis of the response of this gene to abiotic stresses].

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Ingia / Ingia

A cDNA clone containing a fructose-1,6-bisphosphate aldolase (ALD) gene, designated ClAldC, was isolated from a medicinal plant Codonopsis lanceolata. ClAldC is predicted to encode a precursor protein of 358 amino acid residues, and its sequence shares high degrees of homology with a number of other

New molecular insights into osteosarcoma targeted therapy.

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Ingia / Ingia

OBJECTIVE Recent translational studies in osteosarcoma are discussed with the purpose to shed light on the new molecular therapeutic targets. RESULTS The genetic aberrations of vascular endothelial growth factor (VEGF), mammalian target of rapamycin, Wnt signaling pathway, the inactivation of p53,

HIF-driven SF3B1 induces KHK-C to enforce fructolysis and heart disease.

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Ingia / Ingia

Fructose is a major component of dietary sugar and its overconsumption exacerbates key pathological features of metabolic syndrome. The central fructose-metabolising enzyme is ketohexokinase (KHK), which exists in two isoforms: KHK-A and KHK-C, generated through mutually exclusive alternative

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Herbal & Natural Medicine

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