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cyanosis/hemorrhage

Kiungo kimehifadhiwa kwenye clipboard
Ukurasa 1 kutoka 322 matokeo
OBJECTIVE Platelet dysfunction is one of the major haematological disturbances of cardiopulmonary bypass (CPB). In addition, cyanosis is known to cause further coagulation disturbances. METHODS We prospectively studied 110 children under 1 year of age for the effects of cyanosis on baseline platelet

Diagnosis and treatment of hereditary hemorrhagic telangiectasia in a pediatric patient with chronic cyanosis.

Watumiaji waliosajiliwa tu ndio wanaweza kutafsiri nakala
Ingia / Ingia

Telangiectasis of the lungs, with two case reports of hereditary haemorrhagic telangiectasia with cyanosis.

Watumiaji waliosajiliwa tu ndio wanaweza kutafsiri nakala
Ingia / Ingia

Progressive cyanosis in a child with hereditary hemorrhagic telangiectasia (Osler-Weber-Rendu disease).

Watumiaji waliosajiliwa tu ndio wanaweza kutafsiri nakala
Ingia / Ingia

Clinical pathologic conference no. 486 cardiac arrest, peripheral cyanosis, widespread bleeding, thrombocytopenia.

Watumiaji waliosajiliwa tu ndio wanaweza kutafsiri nakala
Ingia / Ingia

Hepatogenous cyanosis and subarachnoid haemorrhage.

Watumiaji waliosajiliwa tu ndio wanaweza kutafsiri nakala
Ingia / Ingia

Upper GI bleeding among neonates admitted to Mulago Hospital, Kampala, Uganda: a prospective cohort study.

Watumiaji waliosajiliwa tu ndio wanaweza kutafsiri nakala
Ingia / Ingia
BACKGROUND The World Health Organization (WHO) reports estimate that 85% of newborn deaths are due to infections, prematurity and fetal distress. These conditions are risk factors for upper GI bleeding (UGIB) in sick neonates. UGIB is associated with poor neonatal outcomes such as prolonged

[Hereditary hemorrhagic telangiectasia: a report of two cases]

Watumiaji waliosajiliwa tu ndio wanaweza kutafsiri nakala
Ingia / Ingia
This article reports two children with hereditary hemorrhagic telangiectasia (HHT). Patient 1 was a boy aged 12 years and was admitted due to intermittent cough and wheezing for more than 10 years. This boy and his mother and grandmother had a history of epistaxis. The boy had a history of the

Perthes Syndrome associated with intramedullary spinal cord hemorrhage in a 4-year-old child: a case report.

Watumiaji waliosajiliwa tu ndio wanaweza kutafsiri nakala
Ingia / Ingia
BACKGROUND Perthes Syndrome (Traumatic asphyxia) is rare, which is caused by sudden compressive chest trauma and characterized by subconjunctival hemorrhage, facial edema, craniocervical cyanosis, and petechiae on the upper chest and face. METHODS We present the case of a 4-year-old Caucasian girl
OBJECTIVE To observe the phenomenon of pulmonary hemorrhage (PH) induced by exogenous endothelin-1 (exET-1) and the antagonizing effect of exogenous calcitonin gene-related peptide (exCGRP) in newborn rats. METHODS (1) To study the exET-1 induced PH: 100 newborn Wistar rats were randomly assigned

Hereditary hemorrhagic telangiectasia or Rendu-Osler-Weber syndrome in the same family.

Watumiaji waliosajiliwa tu ndio wanaweza kutafsiri nakala
Ingia / Ingia
The authors present the case of three patients from the same family in whom hereditary hemorrhagic telangiectasia (HHT) or Rendu-Osler-Weber syndrome was diagnosed. The disease is rare and occurs with multiple telangiectases of the skin and mucosa, and pulmonary arteriovenous fistulae. The clinical

[Value of cyanosis and petechiae in strangulation and concurrent severe anemia (author's transl)].

Watumiaji waliosajiliwa tu ndio wanaweza kutafsiri nakala
Ingia / Ingia
Cyanosis, petechiae, and hemorrhages into the soft tissues and muscles of the neck may be absent after strangulation (throttling) during life, if severe bleeding has happened before. If in severe anemia signs directly or indirectly caused by strangulation have developed, it may be suggested that

[Neonatal varicella: report of a case of bronchopneumonia and hemorrhagic pulmonary edema].

Watumiaji waliosajiliwa tu ndio wanaweza kutafsiri nakala
Ingia / Ingia
We describe the case of a male newborn infant, whose mother developed varicella 20 days before delivery. At 2 hours of life the infant's general state suddenly deteriorated and he presented cyanosis, respiratory distress and emission of pink foam in his mouth. The infant was diagnosed with bilateral

Vitamin K deficiency--late onset intracranial haemorrhage.

Watumiaji waliosajiliwa tu ndio wanaweza kutafsiri nakala
Ingia / Ingia
A retrospective study is presented of the clinical features and outcome of late onset haemorrhagic disease due to vitamin K deficiency in 11 babies who were admitted to the emergency or child neurology unit during a 4-year period (January 1994-December 1997). The disease occurred in infants between
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