l asparaginase/kiharusi

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Neurosurgical management of L-asparaginase induced haemorrhagic stroke.

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Ingia / Ingia

The authors describe a case of L-asparaginase induced intracranial thrombosis and subsequent haemorrhage in a newly diagnosed 30-year-old man with acute lymphoblastic leukaemia who was successfully managed by surgical intervention. At presentation, he had a Glasgow Coma Score of 7/15, was aphasic

Recurrent cerebrovascular accident with L-asparaginase rechallenge.

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Ingia / Ingia

We report a 15-year-old boy diagnosed with acute lymphoblastic leukemia (ALL) in 1983. Induction therapy included L-asparaginase. After the second dose of L-asparaginase, he had a left sided focal seizure and computed tomography (CT) scan of the head showed a right frontal infarct. No further

L-asparaginase therapy with concomitant cranial venous thrombosis: can MRI help avoiding stroke.

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Ingia / Ingia

OBJECTIVE To prospectively use MRI in the early detection of intracranial sino-venous thrombosis during the L-asparaginase induction therapy of acute leukemia thus preventing the evolution of brain venous infarct. METHODS The study population consisted of seventy patients receiving L-asparaginase

Strokes and the neurotoxicity of l-asparaginase.

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Ingia / Ingia

Stroke due to treatment with L-asparaginase in an adult.

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Ingia / Ingia

99Tcm-HMPAO SPECT and magnetic resonance studies in L-asparaginase induced cerebrovascular accident.

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Ingia / Ingia

[Stroke after treatment with L-asparaginase: correlation between clinical improvement, neuroimaging findings, and levels of coagulation factors].

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Ingia / Ingia

Childhood ischemic stroke in a nonurban population.

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Ingia / Ingia

A 10-year, retrospective review of the etiology, outcome, and complications of ischemic stroke in children from a nonurban population was conducted. Twenty-seven children were identified (14 boys, 13 girls), ages 1.25 to 17 years (mean 7.7 years). Etiologies included undetermined (22%), arterial

Intracranial bleeding during therapy with L-asparaginase in childhood acute lymphocytic leukemia.

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Ingia / Ingia

Two patients developed clinical features of intracranial bleeding--which were confirmed by computerized axial tomograms--during their induction therapy for acute lymphocytic leukemia. Coagulation studies showed clotting abnormalities including severe hypofibrinogenemia. These findings most probably

Thrombotic and hemorrhagic strokes complicating early therapy for childhood acute lymphoblastic leukemia.

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Ingia / Ingia

Sudden cerebrovascular insults occurred during or immediately following remission induction therapy in 4 children with acute lymphoblastic leukemia. In 3, cerebral infarction was due to thrombosis. In the fourth, an intracerebral hematoma developed representing either frank hemorrhaging or a

Thromboembolic complications associated with L-asparaginase therapy. Etiologic role of low antithrombin III and plasminogen levels and therapeutic correction by fresh frozen plasma.

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Ingia / Ingia

A case of an 18-year-old woman with acute lymphoblastic leukemia who developed L-asparaginase-associated stroke and subclavian vein thrombosis is presented. The latter was also associated with a Hickman central venous catheter. Thrombotic complications occurred when plasma levels of plasminogen and

The role of drugs in the etiology of stroke.

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Ingia / Ingia

Drugs of many classes have been implicated in hemorrhagic and ischemic stroke. Alcohol in moderation may have a protective effect although in greater doses may predispose to stroke. Drugs such as cocaine, amphetamines and heroin have been associated with stroke by a number of mechanisms.

Cerebrovascular accidents in children with cancer.

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Ingia / Ingia

During a 4-year period, 26 children with systemic malignancies suffered cerebrovascular accidents. These occurred in 17 patients with lymphoreticular malignancy and nine patients with solid tumors. They were the presenting signs of malignancy in three patients and were the direct cause of death in

Neurological complications of chemotherapy to the central nervous system.

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Ingia / Ingia

One of the most common complications of chemotherapeutic drugs is toxicity to the central nervous system (CNS). This toxicity can manifest in many ways, including encephalopathy syndromes and confusional states, seizure activity, headache, cerebrovascular complications and stroke, visual loss,

Cerebrovascular complications in cancer patients.

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Ingia / Ingia

Coagulation disorders are common in cancer patients. In patients with solid tumors, a low-grade activated coagulation can result in systemic and cerebral arterial or venous thrombosis. Cancer treatments may also contribute to this coagulopathy, which usually, but not exclusively, occurs in the

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