myoclonus/infarction

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Ukurasa 1 kutoka 75 matokeo

Unilateral epileptic negative myoclonus following focal lesion of the postcentral cerebral cortex due to acute middle cerebral infarction.

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Ingia / Ingia

Here we report a patient who suffered an acute infarction of the contralateral postcentral cerebral cortex and subsequently developed unilateral partial epilepsy with negative myoclonus. The findings of brain magnetic resonance imaging, polygraphic recordings of the postcentral somatosensory area,

An unusual concomitant tremor and myoclonus after a contralateral infarct at thalamus and subthalamic nucleus.

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Ingia / Ingia

A 72-year-old woman experienced a sudden onset of spontaneous tremor and myoclonus of right extremities that completely subsided 24 hours after onset. Neuroimaging study revealed an infarct at the left ventral portion of thalamus and subthalamic nucleus. Concomitant dyskinetic movement disorders

[So-called laryngeal nystagmus. Progressive myoclonus as a symptom of a cerebellar and brain stem infarct].

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Ingia / Ingia

The present article describes a patient with ischemia in the vascular territory of the posterior inferior cerebellar artery, who developed in addition to other typical signs of the dorsolateral medulla oblongata, myoclonic movements of the ipsilateral vocal cord. Within a period of one year, the

Palatal myoclonus in a patient with a lateral thalamic infarction.

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Ingia / Ingia

Multifocal Myoclonus as a Manifestation of Acute Cerebral Infarction Recovered by Carotid Arterial Stenting.

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Ingia / Ingia

[Unilateral myoclonus induced by hypocalcemia in a case of old cerebral infarction].

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Ingia / Ingia

Serotonin responsive and nonresponsive diurnal depressive mood disorders and pathological affect in thalamic infarct associated with myoclonus and blepharospasm.

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Ingia / Ingia

Palatal myoclonus--a case report.

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Ingia / Ingia

Palatal myoclonus is usually due to a brainstem or cerebellar lesion disrupting the dentato-rubro-olivary pathway. Rarely it may be caused by a cortical lesion. The precipitating factor in 70% of all cases is an infarct. We describe an unusual case of a patient with palatal myoclonus who had an old

Wernekink commissure syndrome with palatal myoclonus at onset: a case report and review of the literature.

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Ingia / Ingia

BACKGROUND Wernekink commissure syndrome causes a peculiar combination of internuclear ophthalmoplegia, dysarthria, and delayed-onset palatal myoclonus. Palatal myoclonus is thought to be secondary to delayed hypertrophic degeneration of the bilateral inferior olivary nuclei secondary to involvement

Wernekink Commissure Syndrome Secondary to Bilateral Caudal Paramedian Midbrain Infarction Presenting with a Unique "Heart or V" Appearance Sign: Case Report and Review of the Literature.

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Ingia / Ingia

Wernekink commissure syndrome secondary to caudal paramedian midbrain infarction (CPMI) is a rare midbrain syndrome involving the decussation of the superior cerebellar peduncle in the caudal paramedian midbrain tegmentum. The central characteristics are constant bilateral cerebellar dysfunction,

[Unilateral palatal myoclonus with peculiar ocular movements--neurotological studies and MRI].

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Ingia / Ingia

This is the first report of unilateral palatal myoclonus with which two different ocular movements were synchronized. A 55-year-old woman was admitted to our hospital due to intubation and dysarthria of sudden onset after three similar attacks for these four years. On admission right ptosis,

Prognostic value of myoclonus status in comatose survivors of cardiac arrest.

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Ingia / Ingia

Generalized myoclonus status is common in comatose patients after cardiac resuscitation, but its prognostic value is uncertain. We studied the clinical, radiologic, and pathologic findings in 107 consecutive patients who remained comatose after cardiac resuscitation. Myoclonus status was present in

Complex movement disorders following bilateral paramedian thalamic and bilateral cerebellar infarcts.

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Ingia / Ingia

Complex movement disorders (CMD; including tremor, dystonias, choreoatheosis, and myoclonus) following infarcts in the posterior and posterolateral thalamic nuclei have been reported. This case of a 59-year-old man who developed CMD following bilateral paramedian and bilateral cerebellar infarcts

Benign segmental myoclonus: electrophysiological evidence of transient dysfunction in the brainstem.

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Ingia / Ingia

We present a 66-year-old patient with segmental myoclonus evoked by a brainstem infarction. The myoclonus appeared soon after a cerebrovascular accident and it was evident in the soft palate, jaw, neck, shoulders and upper limbs. Brain MRI showed infarction in the left pons and left cerebellum.

[Multi-infarct disorder presenting as corticobasal degeneration (DCB): vascular pseudo-corticobasal degeneration?].

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Ingia / Ingia

We report on five patients with a clinical presentation of corticobasal degeneration (CBD), including gradually progressive, asymmetric, L-DOPA-resistant parkinsonism associated variously with apraxia, focal action myoclonus, focal dystonia, cortical sensory loss and alien limb phenomenon. Some

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