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oxalic/infarction

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NakalaMajaribio ya klinikiHati miliki
7 matokeo

[Changes of glutamic-oxalic transaminase in myocardial infarct and stenocardia].

Watumiaji waliosajiliwa tu ndio wanaweza kutafsiri nakala
Ingia / Ingia

[Determination of serum glutamic oxalic acid transaminase in myocardial infarct].

Watumiaji waliosajiliwa tu ndio wanaweza kutafsiri nakala
Ingia / Ingia

[On increase of glutamic-oxalic transaminase (SGPT) activity in myocardial infarction].

Watumiaji waliosajiliwa tu ndio wanaweza kutafsiri nakala
Ingia / Ingia

[Value of phosphohexose isomerase (PHI) determination in myocardial infarct].

Watumiaji waliosajiliwa tu ndio wanaweza kutafsiri nakala
Ingia / Ingia
The biochemical diagnosis of myocardial infarction is based mainly on the enzyme levels of glutamino-oxalic transaminase (SGOT), lactic dehydrogenase (LDH), and creatine-phosphokinase (CPK). The authors have compared the level of phospho-hexo-isomerase (PHI) with the levels of the three other

Primary hyperoxaluria: report of a patient with livedo reticularis and digital infarcts.

Watumiaji waliosajiliwa tu ndio wanaweza kutafsiri nakala
Ingia / Ingia
Primary hyperoxaluria encompasses 3 rare genetic disorders of glyoxylate metabolism characterized by excessive urinary excretion of oxalic acid, resulting in oxalosis. Patients typically have recurrent calcium oxalate nephrolithiasis and nephrocalcinosis, leading to chronic renal failure and death
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