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centratherum anthelminticum/antifungaller

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Background. Opportunistic fungal infections like candidiasis are common in the oral cavity. In recent years Candida species have shown resistance against a number of synthetic drugs. This study assessed the antifungal activity of Centratherum anthelminticum and Ocimum sanctum seed oils against six
BACKGROUND The colonization of Candida albicans on soft liners causes the deterioration of material surface properties and denture stomatitis. OBJECTIVE The purpose of this in vitro study was to investigate the effect of incorporating seed oil on the antifungal properties, surface roughness,

Phytochemical investigation and antifungal activity of the seeds of Centratherum anthelminticum Kuntze.

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Vernodalol mediates antitumor effects in acute promyelocytic leukemia cells.

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Acute promyelocytic leukemia (APL) remains a challenge to cure due to the side effects of cytotoxic chemotherapy and drug resistance. The present study demonstrated that vernodalol, an active compound isolated from Centratherum anthelminticum, suppresses APL cell proliferation and induces cell cycle
BACKGROUND Centratherum anthelminticum (L.) Kuntze (scientific synonyms: Vernonia anthelmintica; black cumin) is one of the ingredients of an Ayurvedic preparation, called "Kayakalp", commonly applied to treat skin disorders in India and Southeast Asia. Despite its well known anti-inflammatory

In silico and In vitro evaluation of the anti-inflammatory potential of Centratherum punctatum Cass-A.

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Centratherum punctatum Cass., a herb belonging to the family Asteraceae has been traditionally used as a curative against diverse disorders like inflammation, tumor, depression, and hypertension. Though the medicinal properties of this plant have been attributed to the presence of flavonoids,
BACKGROUND Centratherum anthelminticum (L.) seeds (CA) is a well known medicinal herb in Indian sub-continent. We recently reported anti-oxidant property of chloroform fraction of Centratherum anthelminticum (L.) seeds (CACF) by inhibiting tumor necrosis factor-α (TNF-α)-induced growth of human
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