ethylene/seizures

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Ethylene glycol: an estimate of tolerable levels of exposure based on a review of animal and human data.

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Upon ingestion ethylene glycol (EG, monoethylene glycol) is rapidly absorbed from the gastrointestinal tract, and depending on the severity of exposure signs of toxicity may progress through three stages. Neurological effects characterize the first step consisting of central nervous depression

The intracerebral administration of phenytoin using controlled-release polymers reduces experimental seizures in rats.

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OBJECTIVE An alternative strategy for the treatment of intractable seizures may be to administer anticonvulsants directly into the brain near the site of a seizure focus using controlled-release polymers. We describe the pharmacokinetics of a phenytoin-ethylene-vinyl acetate (EVAc)

Reversibility of Severe Cerebral Magnetic Resonance Imaging Changes Associated with Ethylene Glycol Toxicity.

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Ethylene glycol (EG), commonly found in antifreeze and deicing solutions, continues to be a cause of morbidity and mortality in the pediatric population. EG metabolism produces toxins that cause metabolic acidosis and calcium oxalate deposition throughout the body. Deposition in the central nervous

Ethylene glycol action on neurons and its cholinomimetic effects.

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Ethylene glycol (EG) is the most representative of the glycols. It is a compound used as painting and plastic solvent, as antifreeze, and in dyes and synthetic fibers. It may also appear as a wine pollutant. Due to these various uses and conditions, EG can produce intoxication in men and animals.

Two gaps too many, three clues too few? Do elevated osmolal and anion gaps with crystalluria always mean ethylene glycol poisoning?

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A 60-year-old African-American man with a medical history significant for heavy alcohol abuse, hypertension, delirium tremens, nephrolithiasis and seizure disorder was brought to the hospital with altered mental status. He was found to have high anion gap metabolic acidosis with significantly

Ethylene glycol toxicosis in a pygmy goat.

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Ingestion of ethylene glycol was responsible for severe azotemia, acidosis, and abnormal anionic gap value in a pygmy goat. Clinical signs consisted of ataxia, polydipsia, decreased rumen motility, and constipation. Nervous signs included depression, absence of menace response, vertical nystagmus,

Acute ethylene oxide intoxication.

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A case of acute ethylene oxide intoxication is described in which neurological involvement was present. Three recurrent seizures over a period of an hour were observed in a 43-year-old female. The patient continued to complain of malaise and inability to perform minor motor tasks up to one week

Ethylene glycol toxicosis in chickens.

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Ethylene glycol (EG) toxicosis was diagnosed on postmortem examination in a white leghorn chicken. The clinical signs were sudden onset of depression, ataxia, convulsions and death. Natural cases of EG toxicosis have not been reported in chickens. This report highlights the importance of including

Transarterial embolization of intracranial dural arteriovenous fistulas with direct cortical venous drainage using ethylene vinyl alcohol copolymer (Onyx).

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OBJECTIVE This communication concerns the new possibilities and technical aspects of using ethylene vinyl alcohol copolymer (Onyx, EVAC) for endovascular treatment of intracranial dural arteriovenous fistulas with direct cortical venous drainage (DAVF-CVs). METHODS Five patients with symptomatic

Does Tc-99m ECD ictal brain SPECT have incremental value in localization of epileptogenic zone and predicting postoperative seizure freedom in cases with discordant video electroencephalogram and MRI findings?

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Objective: Localization of epileptogenic focus in drug-refractory epilepsy using Tc-99m ethylene cystine dimer (ECD) brain single photon emission computed tomography (SPECT) is less studied in patients with discordant findings on video

Does Tc-99m ECD ictal brain SPECT have incremental value in localization of epileptogenic zone and predicting postoperative seizure freedom in cases with discordant video electroencephalogram and MRI findings?

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[Human neurotoxicity of ethylene-bis-dithiocarbamates (EBDC)].

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Ethylene-bis-dithiocarbamates (EBDC) (maneb, mancozeb,...) are fungicides which rarely cause acute toxicity reactions, but may have a severe long-term toxic effect. Twelve cases reported to the Bordeaux Anti-Poison Center over a 10-year period generally exhibited short-term neurological symptoms of

Delayed neurological sequelae from ethylene glycol, diethylene glycol and methanol poisonings.

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BACKGROUND Ethylene glycol, diethylene glycol and methanol are widely available chemicals and are found in a variety of common household products including antifreeze, windshield washer fluid, brake fluid and lubricants. Following ingestion of these glycols and methanol, patients frequently develop

[Acute ethylene glycol poisoning with the course resembling the development of intracranial hematoma].

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Acute poisoning with ethylene glycol manifests itself often with symptoms of central nervous system involvement. The predominating manifestations include consciousness disturbances, seizures, while focal brain damage signs are less frequent. The reported case was hospitalized after trauma to the

Ethylene glycol toxicosis in a free-ranging raccoon (Procyon lotor) from Prince Edward Island.

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A juvenile free-ranging raccoon (Procyon lotor) was presented for acute onset of abnormal mentation and seizures. Ethylene glycol toxicosis was diagnosed on postmortem examination. This report highlights the importance of including ethylene glycol toxicosis on the list of differential diagnoses for

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