aminophylline/hypoxia

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[Effect of Shen-Mai injection and aminophylline on diaphragmatic muscle cell apoptosis and related gene expression in rats with chronic hypoxia].

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OBJECTIVE To investigate the effect of Shen-Mai injection (SMI) and aminophylline on diaphragmatic muscle cell apoptosis and the Fas/FasL expression in chronic hypoxic rats. METHODS Seventy-five male Wistar rats were randomly divided into three equal groups, control group (A group), SMI group (B

Carotid bodies and ventilatory response to hypoxia in aminophylline-treated piglets.

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Peripheral chemoreceptors may be immature in neonatal animals, exhibiting maturational changes in the perinatal period. Even though methylxanthines are respiratory stimulants, many premature neonates do not respond to them. Thus, we hypothesized that carotid body activity is necessary for

Aminophylline increases parasternal intercostal muscle activity during hypoxia in humans.

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To clarify the mechanism of action of aminophylline on the hypoxic ventilatory response in humans, we analyzed the effects of aminophylline on respiratory neural output. To evaluate the respiratory neural output, we analyzed the electromyogram (EMG) of the parasternal intercostal muscle, one of the

Dual effect of aminophylline on the ventilatory response to hypoxia in the rat.

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Male Hooded Wistar rats were exposed to three five-minute periods of hypoxia in which they breathed a gas mixture comprising 7% O2 and 93% N2. Before the second and third hypoxic exposures rats were injected (i.m.) with aminophylline (an adenosine antagonist) at a dose of 15 mg.kg-1. In control

Aminophylline alters the core temperature response to acute hypoxemia in newborn and older guinea pigs.

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In newborns and adults of a number of species, exposure to acute hypoxemia produces a "regulated" decrease in core temperature, the mechanism of which is unknown. The present experiments were carried out on chronically instrumented newborn (5-10 days of age; n = 27) and older (25-30 days of age; n =

Aminophylline effects on ventilatory response to hypoxia and hyperoxia in normal adults.

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In 10 normal young adults, ventilation was evaluated with and without pretreatment with aminophylline, an adenosine blocker, while they breathed pure O2 1) after breathing room air and 2) after 25 min of isocapnic hypoxia (arterial O2 saturation 80%). With and without aminophylline, 5 min of

Coronary vasodilator responses to hypoxia before and after aminophylline.

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1. In previous studies adenosine has been postulated to be the mediator in coronary blood flow regulation and aminophylline was found to inhibit the coronary vasodilator action of adenosine. The present study was performed to determine whether aminophylline inhibits coronary vasodilatation induced

Aminophylline partially blocks ventilatory depression with hypoxia in the awake cat.

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In humans and cats, the ventilatory response to 30 min of moderate hypoxia is biphasic, an initial increase being followed by a decrease in ventilation to levels that are often less than halfway between the initial response and the air-breathing control level. The decrease, or hypoxic depression, is

Effects of aminophylline on hypoxemia-induced ventilatory depression in the cat.

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We designed experiments to evaluate changes in ventral medullary (VM) extracellular fluid (ECF) PCO2 and pH during hypoxemia-induced ventilatory depression (VD). Our aim was to investigate effects of aminophylline on VD and VM ECF acid-base variables. We used aminophylline because it inhibits

Methazolamide Plus Aminophylline Abrogates Hypoxia-Mediated Endurance Exercise Impairment.

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In hypoxia, endurance exercise performance is diminished; pharmacotherapy may abrogate this performance deficit. Based on positive outcomes in preclinical trials, we hypothesized that oral administration of methazolamide, a carbonic anhydrase inhibitor, aminophylline, a nonselective adenosine

Ventilatory response to sustained hypoxia after pretreatment with aminophylline.

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During sustained hypoxia the decline in ventilation that occurs in normal adult humans may be related to central accumulation of a neurochemical with net inhibitory effect. Recent investigations have shown that the putative neurotransmitter adenosine can effect a prolonged respiratory inhibition.

Safety and Ergogenic Properties of Combined Aminophylline and Ambrisentan in Hypoxia.

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We hypothesized that concomitant pharmacological inhibition of the endothelin and adenosine pathway is safe and improves exercise performance in hypoxic humans, via a mechanism that does not involve augmentation of blood oxygenation. To test this hypothesis, we established safety and drug

Anti-hypoxia and anti-oxidation effects of aminophylline on human with acute high-altitude exposure.

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OBJECTIVE To investigate the anti-hypoxia and anti-oxidation effects of aminophylline on human with acute high-altitude exposure. METHODS Totally 100 young male army members newly recruited from Sichuan province (400 meters above sea level) were enrolled. They were randomly divided into two groups:

Does aminophylline improve nocturnal hypoxia in patients with chronic airflow obstruction?

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The progression of pulmonary hypertension secondary to chronic airflow obstruction is thought to be related to the degree of nocturnal oxygen desaturation. We have studied 11 patients with severe smoking-related hypoxic chronic airflow obstruction (mean FEV1 0.67 L, mean arterial PO2 6.83 kPa) who

Effect of aminophylline and relief from hypoxia on central sleep apnoea due to medullary damage.

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A 17-year old boy presented with severe, predominantly central sleep apnoeas secondary to structural damage in the medulla. At low O2 saturation, the electroencephalogram showed the sudden onset of slow waves. Hypercapnic ventilatory response was low and hypoxic ventilatory response was absent. Low

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