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atropine/accidente cerebrovascular
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Does intravenous atropine affect stroke volume variation in man?
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OBJECTIVE
Currently there are no reports of the effect of increasing heart rate (HR) induced by intravenous atropine on stroke volume variation (SVV). We hypothesized that increasing HR alters the value of SVV. This prospective study aimed to investigate changes in SVV values by increasing HR
Stroke volume changes during dobutamine-atropine stress echocardiography: the influence of heart rate and ischaemia.
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BACKGROUND
A decrease in stroke volume during dobutamine-atropine stress echocardiography heralds ischaemia and possible hypotension. Hypotension results from worsening of LV-function (as a result of ischaemia) left ventricular outflow tract obstruction or hypovolemia, while an increase of stroke
[Severe atropine poisoning mimicking acute stroke].
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BACKGROUND
Atropine is a strong antagonist of muscarinic receptors widely used in various diseases because of its anticholinergic action.
METHODS
We report here a case of accidental poisoning due to ingestion of atropine eyes drops that caused severe neurologic disorders mimicking an acute stroke.
Atropine and the control of heat stroke.
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Changes in cardiac output, stroke volume, and central venous pressure induced by atropine in man.
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[Cardiac output, heart rate and stroke volume, blood pressure and peripheral circulatory resistance in dogs under the influence of physical work and the administration of catecholamines and atropine].
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Normal stroke volume and cardiac output response during dobutamine stress echocardiography in subjects without left ventricular wall motion abnormalities.
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Dobutamine stress echocardiography has become widely utilized for evaluation of coronary artery disease, but the expected responses of stroke volume and cardiac output to the high doses of dobutamine administered in these studies are not known. To determine these responses, stroke volume and cardiac
Filling and arterial pressures as determinants of RV stroke volume in the sheep fetus.
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Right ventricular (RV) function was investigated in nine fetal lambs (125-130 days gestation) that were instrumented with pulmonary artery electromagnetic flow sensors and vascular catheters. Control arterial CO2 and O2 tension, pH, and hematocrit values were 46.1 +/- 1.6 (SD) Torr, 20.6 +/- 1.8
Middle cerebral artery occlusion in Macaca fascicularis: acute and chronic stroke evolution.
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BACKGROUND
An intravascular stroke model designed for magnetic resonance imaging was developed in Macaca fascicularis (M. fascicularis) to characterize serial stroke lesion evolution. This model produces a range of stroke lesion sizes which closely mimics human stroke evolution. This paper describes
Baroreflex control of stroke volume in man: an effect mediated by the vagus.
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1. Beat-by-beat changes in cardiac performance in response to arterial baroreceptor stimulation induced by phenylephrine were evaluated by pulsed-wave aortic Doppler ultrasound in eighteen subjects. Stroke distance was used as an index of stroke volume and minute distance as an index of cardiac
Sinus node suppression in acute strokes--case reports.
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The authors report on 5 patients, 4 of them observed over a period of only two months in one community hospital, who developed profound sinus node suppression in the early phase following acute strokes. This complication resulted in the death of 2; in the remaining 3, it was controlled by electrical
[Effects of atropine in coronary heart disease].
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For the detection of atropine effects the hemodynamic changes of 50 patients with coronary heart disease being premedicated intramuscularly with pethidine/diazepam have been registered 3 and 6 minutes after intravenous atropine application (0.01 mg/kg). All patients showed an increase of the heart
[Cardiovascular changes caused by atropine in normo- and hypothermic methoxyflurane anaesthesia (author's transl)].
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The authors examined in 25 patients, of which 15 were normo- and 10 in hypothermic, the cardiovascular effects of 1 mg atropine i.v. In normothermia the heart rate increased significantly from 100 to 110 beats/min after atropine. At the same time the stroke index and stroke work decreased
Cardiovascular response to fentanyl-droperidol and atropine in the dog.
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A combination of fentanyl-droperidol was administered intravenously alone or with atropine sulfate (2 doses--0.04 or 0.02 mg/kg of body weight) to determine if stable neuroleptanalgesia could be produced in the dog. Cardiovascular responses were recorded at 5, 15, and 30 minutes. Fentanyl-droperidol
The QT interval and cycle length: the influence of atropine, hyoscine and exercise.
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Twenty-seven healthy male subjects of mean age 24.3 +/- 4.0 years and mean weight 74.9 +/- 9.1 kg took part in an investigation to assess the most suitable correction for the QT interval as a function of cardiac cycle length. 547 sets of data points were generated. Atropine 0.6, 1.2 and 1.8 mg, and
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