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formaldehyde/carie dentaire
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Removing formaldehyde from embalmed cadavers by percolating the body cavities with dilute ethanol.
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Formaldehyde was removed from embalmed animal cadavers by pumping ethanol (20%) through the pleural and peritoneal cavities of 4 goats, 4 cows and 4 horses. The goats were percolated intermittently for 7 days and the large animals continuously for 72 h. Just after opening the body cavities, samples
Formaldehyde and the risk of squamous cell carcinoma of the sinonasal cavities.
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A study of 759 histologically verified cancers of the nasal cavity (287 cases), paranasal sinuses (179 cases), and nasopharynx (293 cases) and 2465 cancer controls diagnosed in Denmark between 1970 and 1982 was conducted to investigate the importance of occupational exposure to formaldehyde.
Formaldehyde and cancers of the pharynx, sinus and nasal cavity: I. Occupational exposures.
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A population-based case-control study was undertaken in 13 counties of western Washington to determine if occupational formaldehyde exposure was related to cancer of the oro- and hypopharynx (OHPC, N = 205), nasopharynx (NPC, N = 27) or sinus and nasal cavity (SNC, N = 53). Controls were selected by
Sensitive Detection of Ambient Formaldehyde by Incoherent Broadband Cavity Enhanced Absorption Spectroscopy.
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Formaldehyde (HCHO) is the most abundant atmospheric carbonyl compound and plays an important role in the troposphere. However, HCHO detection via traditional incoherent broadband cavity enhanced absorption spectroscopy (IBBCEAS) is limited by short optical path lengths and weak light intensity.
Nasal cavity deposition, histopathology, and cell proliferation after single or repeated formaldehyde exposures in B6C3F1 mice and F-344 rats.
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Inhalation exposure for 2 years to 14.3 ppm formaldehyde (HCHO) induced a 50% incidence of squamous cell carcinoma in the nasal cavity of F-344 rats but only a 3.3% incidence in B6C3F1 mice. Since the response was concentration and species dependent, species differences in nasal cavity "dose" were
Effects of formaldehyde vapor on the nasal cavity and lungs of F-344 rats.
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Histopathological and biochemical examinations of the nasal cavity and lungs of rats inhaling 145.6 ppm (high-dose) or 15.0 ppm (low-dose) formaldehyde vapor for 6 h revealed dose-related damage. The contents of non-protein SH groups (NPSH) in the nasal mucosa and lung, and lipid peroxide (LPO) in
Induction of squamous cell carcinomas of the rat nasal cavity by inhalation exposure to formaldehyde vapor.
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Groups of 120 male and 120 female rats were exposed by inhalation to 0, 2, 6, or 15 ppm formaldehyde vapor 6 hr/day, 5 days/week, for 18 months of a 24-month study. The present communication describes interim findings based on data available after 18 months of exposure. Squamous cell carcinomas
Formaldehyde and cancers of the pharynx, sinus and nasal cavity: II. Residential exposures.
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To investigate the possible association between residential formaldehyde exposures and risk of cancer of the oro- and hypopharynx (OHPC, N = 205), nasopharynx (NPC, N = 27) and sinus and nasal cavity (SNC, N = 53), a population-based case-control investigation was carried out in 13 counties of
Cancer of the nasal cavity and paranasal sinuses, and formaldehyde exposure.
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This case-control study of nasal and paranasal sinus tumors, in males diagnosed between 1978 and 1981 in the Netherlands, was designed to identify environmental risk factors. Special attention was given to assessing any association between nasal cancer and an occupational history of possible
Malignant melanomas of the nasal cavity after occupational exposure to formaldehyde.
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Formaldehyde is a well known nasal carcinogen in rodents, but so far there has been no convincing evidence that workers occupationally exposed to formaldehyde have an increased risk of nasal cancer. In this study three cases of malignant melanoma of the nasal mucosa in persons occupationally exposed
Mortality from solid cancers among workers in formaldehyde industries.
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In industrial workers, formaldehyde exposure has been associated with cancer of the nasal cavities, nasopharynx, prostate, lung, and pancreas; however, these associations are inconsistent and remain controversial. Animals exposed to formaldehyde show excesses of nasal cancer. In an extended
Dual-feedback mid-infrared cavity-enhanced absorption spectroscopy for H2CO detection using a radio-frequency electrically-modulated interband cascade laser.
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A mid-infrared cavity-enhanced sensor system was demonstrated for the detection of formaldehyde (H2CO) using a continuous-wave (cw) interband cascade laser (ICL) centered at 3599 nm. A compact Fabry-Perot (F-P) cavity with a physical size of 38 × 52 × 76 mm3 was developed consisting of two concave
Molecular Changes in the Nasal Cavity after N, N-dimethyl-p-toluidine Exposure.
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N, N-dimethyl-p-toluidine (DMPT; Cas No. 99-97-8), an accelerant for methyl methacrylate monomers in medical devices, is a nasal cavity carcinogen according to a 2-yr cancer study of male and female F344/N rats, with the nasal tumors arising from the transitional cell epithelium. In this study, we
Formaldehyde exposure in a gross anatomy laboratory.
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A gross anatomy laboratory for medical students was evaluated for formaldehyde levels throughout its eight-week term. Results indicated that exposures for students and instructors were below the 3-ppm permissible exposure limit (assuming a maximum of five hours of daily exposure) established by the
The importance of delivered dose in estimating low-dose cancer risk from inhalation exposure to formaldehyde.
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Data have recently been obtained on the concentration of formaldehyde covalently bound to the respiratory mucosal DNA of Fischer-344 rats following two 6-hr inhalation exposures to gaseous formaldehyde. These data provide a direct short-term measure of the delivered formaldehyde dose in target
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