chest pain/infarction

OBJECTIVE Although previous studies reported that late reperfusion might prevent left ventricular dilation after acute myocardial infarction (AMI), implication of persistent ischemic chest pain on admission remains to be investigated. This study was undertaken to assess the implication of persistent

BACKGROUND Recent studies have indicated that it may be safe to discharge chest pain patients with an initial high-sensitivity cardiac troponin T (hs-cTnT) level of <5 ng/L from the emergency department (ED) without further evaluation. We sought to assess the effects of discharge from the ED versus

The best management of chest pain patients who rule out for myocardial infarction (MI) in the high-sensitivity troponin (hsTn) era remains elusive. Patients, especially those with non-low clinical risk scores, are often referred for inpatient ischemic testing to uncover obstructive coronary artery

BACKGROUND The Universal Definition of Myocardial Infarction incorporates elevated cardiac troponin levels (> 99th percentile) together with a significant rise/fall of troponins as biochemical criterion. We sought to evaluate the clinical implications of the relative change of cardiac troponin I

In 422 patients admitted from the emergency department (ED) for suspected acute myocardial infarction, the hypothesis that chest pain that persists on arrival in the ED or recurs during the initial ED evaluation is a useful predictor of acute myocardial infarction (AMI) and complications of coronary

The contribution of electrocardiograms, serum enzymes and history of chest pain to the diagnosis of acute myocardial infarction (AMI) was examined in a series of 3123 persons with a definite acute myocardial infarction registered in a community-based myocardial infarction register study in North

BACKGROUND Patients who are hospitalized because of chest pain and suspected acute myocardial infarction, but in whom the diagnosis is ruled out, are at high risk for subsequent cardiac events (cardiac death or nonfatal acute myocardial infarction). Risk stratification was done for 158 such patients

Advances over the past few years have led to the use of hand-held point-of-care diagnostic tests to expedite testing for cardiac enzymes indicative of acute myocardial infarction. Although cardiac enzymes such as troponin I and CKMB are reliable markers of acute myocardial infarction, they cannot be

Plasma magnesium concentrations were monitored daily in 86 patients who were admitted to a coronary care unit with a provisional diagnosis of acute myocardial infarction. Twenty-six patients had suffered a myocardial infarction, while the remainder had angina or non-cardiac chest pain. Magnesium

OBJECTIVE To assess whether the admission of patients with chest pain to a stepdown unit would jeopardize the outcome of those patients who ultimately "ruled in" for a myocardial infarction. METHODS We compared the risk of an adverse outcome in initially uncomplicated, "rule-out myocardial

In half of the patients admitted with chest pain on suspicion of an acute myocardial infarction (AMI), this diagnosis is not confirmed (non-AMI). Both AMI and non-AMI patients have a mortality which exceeds the mortality of the background population in the years following discharge based on a high

This paper describes 109 patients who had their first myocardial infarction and were then followed up for 3 to 8 years. The following data were collected at the time of the infarction: duration and severity of chest pain, type of infarction and peak SGOT (serum glutamic oxaloacetic transaminase)

Patients presenting to the emergency department with chest pain are triaged to early reperfusion therapies based on their initial 12-lead electrocardiogram (ECG). The standard 12-lead ECG lacks sensitivity to detect acute myocardial infarction (AMI). Electrocardiographic diagnosis of

BACKGROUND Treatment of patients with acute ST elevation myocardial infarction starts after the onset of chest pain, involves contacts with medical services, aimed at attempting to recanalize the infarct-related artery with primary percutaneous coronary intervention. True ischaemic time correlates